Apoptosis, Pyroptosis, and Necrosis: Mechanistic Description of Dead and Dying Eukaryotic Cells
By S. Fink, B. Cookson. Infection and Immunity, 2005
2,137 citations recorded by the source corpus.
Subfields: Epidemiology, Molecular Biology, Parasitology
View Paper: https://www.semanticscholar.org/paper/748096c31f2d9246fde3d01152864cefef398929
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In Epidemiology
- Fabio Martinon
- J. Tschopp
- Annick Mayor
- R. Medzhitov
- Rebecca S. Y. Wong
- Edward A. Miao
- B. Cookson
- S. Fink
In Molecular Biology
- J. Tschopp
- G. Núñez
- T. Kanneganti
- Fabio Martinon
- Kate Schroder
- Edward A. Miao
- L. Franchi
- Annick Mayor
In Parasitology
Citations in the corpus
- Understanding SARS-CoV-2-Mediated Inflammatory Responses: From Mechanisms to Potential Therapeutic Tools (2020, background): “2019), which can cause 70 the release of large amounts of proinflammatory factors (Fink and Cookson, 2005).”
- Oxidized mitochondrial DNA activates the NLRP3 inflammasome during apoptosis. (2012, background): “Recent studies have described a caspase-1-dependent form of programmed cell death called pyroptosis (Fink and Cookson, 2005).”
- Application of biosynthesized silver nanoparticles for the control of land snail Eobania vermiculata and some plant pathogenic fungi (2015, background): “These results are signs of cell death [43].”
- Function of Nod‐like receptors in microbial recognition and host defense (2009, background): “Pyroptosis is typically associated with rapid disruption of cell membrane integrity and the maturation of pro-IL-1b (213, 214).”
- NLRP3 inflammasome activation results in hepatocyte pyroptosis, liver inflammation and fibrosis (2014, background): “Casp1 activation can also directly induce a distinct form of programmed cell death called pyroptosis.(11-12) Although, pyroptosis has been well described in vitro, its relevance in vivo has remained unclear and supported mainly by indirect reports demonstrating Casp1 mediated effects independent of ”
- Adipocyte death defines macrophage localization and function in adipose tissue of obese mice and humans Published, JLR Papers in Press, September 8, 2005. DOI 10.1194/jlr.M500294-JLR200 (2005, background): “, “necrosis-like programmed cell death”, “paraptosis” ) have recently been described in which apoptotic signaling pathways and proteolytic cascades may participate, but the morphology of cell execution is predominantly necrotic (27, 40).”
- Apoptosis in cancer: from pathogenesis to treatment (2011, background): “which are primarily responsible for the initiation of the apoptotic pathway and 2) effector caspases (caspase-3, -6 and -7) which are responsible in the actual cleavage of cellular components during apoptosis [57].”
- Caspase‐1‐dependent pore formation during pyroptosis leads to osmotic lysis of infected host macrophages (2006, background): “4C and G) consistent with maintenance of an intact plasma membrane throughout apoptosis (Fink and Cookson, 2005).”
- Rapid Neutrophil Destruction following Phagocytosis of Staphylococcus aureus (2010, neutral): “Further studies with additional isogenic gene deletion strains are needed to determine whether cytolytic toxins other than PVL contribute to the PMN lysis described herein.Another notable finding was that lysis of PMNs was significantly increased following uptake of serum opsonized of LAC compared w”
- Origin and physiological roles of inflammation (2008, background): “Third, apoptosis induced by infection (including the caspase-1-dependent process, known as pyroptosi”
- Pyroptosis: host cell death and inflammation (2009, background): “Although apoptosis was the first well-recognized programme of eukaryotic cell death, 'programmed cell death' is broadly applied to several endogenous genetically defined pathways in which the cell plays an active part in its own destructio”