The relation of markers of inflammation to the development of glucose disorders in the elderly: the Cardiovascular Health Study.

By J. Barzilay, L. Abraham, S. Heckbert, M. Cushman, Lewis H. Kuller, Helaine E. Resnick, R. Tracy. Diabetes, 2001

603 citations recorded by the source corpus.

Subfields: Endocrinology, Diabetes and Metabolism, Epidemiology, Pulmonary and Respiratory Medicine

View Paper: https://www.semanticscholar.org/paper/745ff03fdf22b04de3b66b55836f62e6973c73ae

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Endocrinology, Diabetes and Metabolism

1. J. Pickup
2. George Davey Smith
3. Anne B. Newman
4. B. K. Pedersen

In Epidemiology

1. J. Pickup
2. A. M. Petersen
3. B. K. Pedersen
4. C. Fischer
5. José Fernández-Real
6. J. Bastard
7. George Davey Smith
8. Anne B. Newman

In Pulmonary and Respiratory Medicine

1. George Davey Smith
2. B. K. Pedersen
3. J. Pickup

Citations in the corpus

• The anti-inflammatory effect of exercise. (2005, background): “Low-grade chronic inflammation is reflected by increased C-reactive protein (CRP) concentrations and increased systemic levels of some cytokines (118) and several reports investigating various markers of inflammation in different population groups have confirmed an association between low-grade syst”
• Mendelian randomization: Using genes as instruments for making causal inferences in epidemiology (2008, positive): “suggested that therapeutics aimed at reducing CRP levels should be developed to prevent and/or treat these conditions [58–63].”
• Insulin resistance and chronic cardiovascular inflammatory syndrome. (2003, positive): “In the Cardiovascular Health Study, however, baseline fibrinogen, white blood cell and platelet counts, albumin, and factor VIIIc were not associated with development of diabetes, whereas elevated baseline CRP anticipated glucose progression even after adjustment for known baseline predictors of fas”
• Brain-derived neurotrophic factor (BDNF) and type 2 diabetes (2007, background): “Systemic low-grade inflammation is defined as two to fourfold increases in circulating levels of inflammatory markers such as CRP [31], and many reports confirm an association between low-grade systemic inflammation on the one hand, and the metabolic syndrome, type 2 diabetes and atherosclerosis on ”
• Recent advances in the relationship between obesity, inflammation, and insulin resistance. (2006, background): “Otherwise, there is at least a two-fold higher risk of type 2 diabetes within 3-4 years in obese individuals with higher CRP levels [27].”
• The metabolic syndrome, inflammation, and risk of cognitive decline. (2004, background): “Exclusion criteria included (1) any difficulty with activities of daily living, (2) clinical dementia (based on Diagnostic and Statistical Manual of Mental Disorders, 4th edition criteria), (3) inability to communicate with the interviewer, (4) intention of moving out of the vicinity in the next yea”
• Inflammation and activated innate immunity in the pathogenesis of type 2 diabetes. (2004, background): “Cardiovascular Health Study (CRP) (6), in Pima Indians (white blood count) (7), for multiethnic subjects in the U.”
• C-reactive protein is an independent predictor of risk for the development of diabetes in the West of Scotland Coronary Prevention Study. (2002, background): “Our data in middle-aged men and data from the abovementioned studies (6,13) in women and the elderly indicate that CRP may be a useful predictor of diabetes.”
• Inflammatory cytokines and the risk to develop type 2 diabetes: results of the prospective population-based European Prospective Investigation into Cancer and Nutrition (EPIC)-Potsdam Study. (2003, result): “Although previous studies demonstrated that inflammatory markers are associated with future type 2 diabetes, even after adjustment for fasting insulin (6,8), it remains to be elucidated whether this holds true for the cytokine changes described here.”