Recent advances in the relationship between obesity, inflammation, and insulin resistance.

By J. Bastard, M. Maachi, C. Lagathu, Min Ji Kim, M. Caron, H. Vidal, J. Capeau, B. Fève. European Cytokine Network, 2006

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Epidemiology of Obesity and Diabetes and Their Cardiovascular Complications (2016, unknown): “comorbidities, such as dyslipidemia, hypertension, glucose intolerance, endothelial dysfunction, and inflammation.(86,87)”
Adipokines: the missing link between insulin resistance and obesity. (2008, background): “Interleukin (IL)-6 is a cytokine produced by several cells fibroblasts, endothelial cells, monocytes) and by adipose tisue, which is increased in obesity [1,2].”
From Monocytes to M1/M2 Macrophages: Phenotypical vs. Functional Differentiation (2014, background): “These are metabolic syndromes that can lead to a switch in the phenotype of adipose tissue macrophages from wound healing (as in healthy non-obese human beings) to classically activated macrophages (200, 201); cancer, where the tumor-infiltrating classically activated macrophages have the potential ”
Obesity and inflammation: the linking mechanism and the complications (2016, background): “Adiponectin is a protein hormone derived from adipocytes that has gained considerable importance due to its positive impacts on inflammation [99], atherosclerosis, T2DM, and insulin resistance [99–101]; thus, it links the adipose tissue directly with the cornerstones of metabolic abnormalities [102]”
An adaptive annotation approach for biomedical entity and relation recognition (2016, unknown): “tissues followed by chronic hyperglycemia) [48–50].”