Resistin / Fizz3 expression in relation to obesity and peroxisome proliferator-activated receptor-gamma action in humans.

By D. Savage, C. Sewter, Ellen S. Klenk, David G. Segal, Antonio Vidal-Puig, R. Considine, Stephen O’Rahilly. Diabetes, 2001

785 citations recorded by the source corpus.

Subfields: Epidemiology, Molecular Biology, Physiology

View Paper: https://www.semanticscholar.org/paper/6c284985852691ff3f187c5cb63c7ae20f35cadf

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Epidemiology

1. K. Walsh
2. G. Steinberg
3. A. Chait
4. Laura J. den Hartigh
5. J. Oakhill
6. S. Galic
7. N. Ouchi
8. K. Frayn

In Molecular Biology

1. K. Walsh
2. Mitchell A. Lazar
3. A. Chait
4. M. Qatanani
5. G. Steinberg
6. Laura J. den Hartigh

In Physiology

1. G. Steinberg
2. M. Qatanani
3. A. Chait
4. Laura J. den Hartigh
5. S. Galic
6. J. Oakhill
7. K. Walsh
8. N. Ouchi

Citations in the corpus

• Adipokines in inflammation and metabolic disease (2011, background): “, whereas in humans, resistin is mainly produced by macrophages and monocytes, and it is not detectable in adipocyte”
• Adipose tissue as an endocrine organ. (2010, background): “The importance of resistin in humans is less clear as not all studies report increases in serum resistin in obese type 2 diabetics (Savage et al., 2001; Sentinelli et al., 2002), however, it should be noted that the −420G/G genotype of the resistin promoter region has been associated with elevated r”
• What We Talk About When We Talk About Fat (2014, neutral): “This relationship has been harder to demonstrate in humans, however, in whom resistin appears to be secreted primarily by circulating monocytes (Savage et al., 2001).”
• Adipose Tissue Distribution, Inflammation and Its Metabolic Consequences, Including Diabetes and Cardiovascular Disease (2020, background): “It was originally described as an adipocyte-specific hormone, but it is now thought to originate from macrophages residing in inflamed adipose tissue in mice (103) and from circulating monocytes and tissue macrophages in humans (104, 105).”
• Adipose tissue as a buffer for daily lipid flux (2002, background): “Furthermore, the initial findings on resistin have been challenged [104, 105].”
• Mechanisms of obesity-associated insulin resistance: many choices on the menu. (2007, background): “While mouse resistin is exclusively expressed in white adipose tissue, human resistin is mainly expressed in circulating mononuclear cells (Savage et al. 2001; Steppan et al. 2001; Patel et al. 2003).”