Astrocyte-neuron metabolic relationships: for better and for worse.
By Igor Allaman, M. Bélanger, Pierre J. Magistretti. Trends in Neurosciences, 2011
657 citations recorded by the source corpus.
Subfields: Neurology, Physiology
View Paper: https://www.semanticscholar.org/paper/6bd63397936be24b26f1c2417d4589134a7e6dd8
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Neurology
In Physiology
- Melissa A Brown
- R. Ransohoff
- Igor Allaman
- Pierre J. Magistretti
- B. Barres
- D. Bailey
- Maria Luisa Fiorello
- Alec C. Kimmelman
Citations in the corpus
- Glial fibrillary acidic protein (GFAP) and the astrocyte intermediate filament system in diseases of the central nervous system. (2015, background): “They are indispensable for proper brain development [5 ,6 ,7 ,8], play fundamental roles in maintaining ion, neurotransmitter, water [9] and energy homeostasis [10], and modulate neuronal signaling [11,12].”
- Astrocytes and disease: a neurodevelopmental perspective. (2012, background): “2009) and hypoxia (Vangeison and Rempe 2009), among others (Allaman et al. 2011).”
- Metabolic Interactions in the Tumor Microenvironment (2017, background): “This model of symbiotic metabolism is reminiscent of the glucose-lactate shuttle observed in the brain, in which glucose-derived lactate derived from astrocytes is shuttled to neurons [28].”
- 13 reasons why the brain is susceptible to oxidative stress (2018, background): “complex and involves neuronal-glia metabolic coupling (reviewed in [3,92]).”
- Innate immunity in the central nervous system. (2012, positive): “In inflammatory conditions, CXCL12 plus TNF-α signal to microglia to produce large quantities of TNF-α .”
- Brain energy metabolism: focus on astrocyte-neuron metabolic cooperation. (2011, background): “…to neurodegenerative processes is currently under intense investigation, and a rapidly growing body of evidence indicates that enhancing or reestablishing astrocytic functions may represent promising therapeutic avenues for neurodegenerative disease (see Allaman et al., 2011, for review).”