Low counts of Faecalibacterium prausnitzii in colitis microbiota

By Harry Sokol, P. Seksik, J. Furet, O. Firmesse, I. Nion‐Larmurier, L. Beaugerie, J. Cosnes, G. Corthier, P. Marteau, J. Doré. Inflammatory Bowel Diseases, 2009

1,262 citations recorded by the source corpus.

Subfields: Genetics, Infectious Diseases, Molecular Biology

View Paper: https://www.semanticscholar.org/paper/65434ac888c90a632a1326970b7006777892e42f

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Genetics

In Infectious Diseases

In Molecular Biology

Citations in the corpus

High-throughput clone library analysis of the mucosa-associated microbiota reveals dysbiosis and differences between inflamed and non-inflamed regions of the intestine in inflammatory bowel disease (2011, background): “Recently, a reduction in Faecalibacterium prausnitzii has been implicated in CD aetiology [31,42].”
Faecalibacterium prausnitzii and human intestinal health. (2013, background): “3) ! [22] UC Mucosal pouch biopsy Sequencing 16S rRNA 8 51 (19–63) # [53] UC Fecal In vitro: M-SHIME 6 40.”
Metagenomic biomarker discovery and explanation (2011, background): “Comparisons between healthy and diseased tissues have highlighted the importance of tasks such as class discovery (detecting novel subtypes of a disease) and class prediction (determining the subtype of a new sample) [1-4], and recent metagenomic assays have shown that human microbial communities ca”
Dysbiosis of the faecal microbiota in patients with Crohn's disease and their unaffected relatives (2011, result): “In agreement with recent findings of Sokol and co-workers, significantly lower numbers of this species were also detected by RT-PCR in patients with CD as compared with unaffected subjects.(33) In contrast, we found that R gnavus appeared more in the predominant faecal microbiota of patients with CD”
Faecalibacterium prausnitzii: from microbiology to diagnostics and prognostics (2017, background): “prausnitzii level recovery has been reported in feces during remission (Swidsinski et al., 2008; Sokol et al., 2009), whereas it has been observed that in mucosa, depletion of this species occurs regardless of patients disease activity status (Willing et al.”
European consensus conference on faecal microbiota transplantation in clinical practice (2017, background): “48 It is plausible that ethanol, beyond killing pathogens, can also severely alter the composition of commensal microbiota, possibly eliminating critical elements such as bacterio-phages, fungi and non-sporulating bacterial components.”
Through Ageing, and Beyond: Gut Microbiota and Inflammatory Status in Seniors and Centenarians (2010, background): “prausnitzii in centenarians is of interest as this species is known to affect the gut inflammation processes, exerting a butyrate-independent anti-inflammatory effect [38,39].”
Connecting dysbiosis, bile-acid dysmetabolism and gut inflammation in inflammatory bowel diseases (2012, neutral): “Purine analogues (%) 1 (2) 1 (2) 2 (5) 2 (5) 5 (12) 7 (17) –”
Bacteroides thetaiotaomicron and Faecalibacterium prausnitzii influence the production of mucus glycans and the development of goblet cells in the colonic epithelium of a gnotobiotic model rodent (2013, background): “Intestinal disorders, such as inflammatory bowel diseases [22-25], irritable bowel syndrome [26], colorectal cancer [27,28] and obesity [29] have all been reported to be associated with a diminished prevalence and abundance of F.”