Adipose tissue-derived factors: impact on health and disease.

By M. Trujillo, P. Scherer. Endocrine reviews, 2006

658 citations recorded by the source corpus.

Subfields: Cardiology and Cardiovascular Medicine, Epidemiology, Physiology

View Paper: https://www.semanticscholar.org/paper/62275b0a19cd2104bf8a8e041735f55f14cbaedc

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Cardiology and Cardiovascular Medicine

1. Haiming Cao
2. M. Cerf
3. P. Calder
4. B. Spiegelman
5. E. Rosen

In Epidemiology

1. Haiming Cao
2. M. Cerf
3. P. Calder
4. S. Fried
5. B. Spiegelman
6. E. Rosen

In Physiology

1. Christopher B. Newgard
2. Haiming Cao
3. M. Cerf
4. Guoyao Wu
5. P. Calder
6. S. Fried
7. D. Muoio
8. Karine Clément

Citations in the corpus

• Dietary factors and low-grade inflammation in relation to overweight and obesity (2011, positive): “these may represent major contributors to the production of adipokines((12,13)).”
• Beta Cell Dysfunction and Insulin Resistance (2013, background): “In addition, the adipokine hormones and proinflammatory cytokines that are produced by adipose tissue can influence insulin signaling via diverse mechanisms (Shoelson et al., 2006; Trujillo and Scherer, 2006) and these processes may interact with genetic variants influencing insulin resistance pathw”
• Glycine metabolism in animals and humans: implications for nutrition and health (2013, background): “This metabolic disorder increases the risk for type II diabetes and premature death (Trujillo and Scherer 2006).”
• Molecular and metabolic mechanisms of insulin resistance and β-cell failure in type 2 diabetes (2008, background): “The ensuing decade of research has revealed that adipose cells also produce other peptide hormones, including adiponectin ( ACRP30 ), retinol-binding protein-4 ( RBP4 ) and resistin , and proinflammatory cytokines such as interleukin (IL)-6 and tumour necrosis factor-α ( TNFα”
• Adipocytokines in Obesity and Metabolic Disease (2014, positive): “…often result in impaired organ communications Adipo Leptin Adipo Resist aP2 …… Fat cell Immune cell Cy tok ine s FF A and metabolic abnormalities in multiple tissues thereby constituting a critical pathological component in the development of metabolic disease (Trujillo & Scherer 2006).”
• Adipose Tissue Heterogeneity: Implication of depot differences in adipose tissue for Obesity Complications (2012, background): “Adipocytes produce several hormones, most notably leptin and adiponectin, which modulate appetite, fuel metabolism, innate immune function, and reproduction (Trujillo and Scherer, 2006).”
• A Branched-Chain Amino Acid-Related Metabolic Signature that Differentiates Obese and Lean Humans and Contributes to Insulin Resistance (2009, background): “Consistent with numerous prior reports (Bjorbaek and Kahn, 2004; Trujillo and Scherer, 2006), the adipocyte-derived hormones leptin and adiponectin were reciprocally altered by obesity, such that leptin levels were 3.”