Immunology of hepatitis B virus and hepatitis C virus infection

By B. Rehermann, M. Nascimbeni. Nature reviews. Immunology, 2005

1,693 citations recorded by the source corpus.

View Paper: https://www.semanticscholar.org/paper/5c9341ea1c0f3bc3386b8b0fbf32e000197cedf8

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Citations in the corpus

Vaccine instability in the cold chain: mechanisms, analysis and formulation strategies. (2014, neutral): “[158,160,161].”
Redefining chronic viral infection. (2009, neutral): “Continuous replication can generate up to 10 particles per day for HBV and HCV (Rehermann and Nascimbeni, 2005).”
Induction of a Striking Systemic Cytokine Cascade prior to Peak Viremia in Acute Human Immunodeficiency Virus Type 1 Infection, in Contrast to More Modest and Delayed Responses in Acute Hepatitis B and C Virus Infections (2009, background): “adults (38), indicating that the rapid induction of a robust”
Hepatocellular carcinoma pathogenesis: from genes to environment (2006, background): “A robust T-cell immune response is presumably elicited to combat viral infection, however, this response contributes to hepatocyte necrosis, inflammation and consequently regeneration, leading to carcinogenesi”
Exhaustion of tumor-specific CD8⁺ T cells in metastases from melanoma patients. (2011, background): “In humans, functional deficits were found in HIV-1–, HCV-, and HBV-specific CD8+ T cells (11, 16, 17), and a recent gene”
Review of hepatocellular carcinoma: Epidemiology, etiology, and carcinogenesis (2017, background): “Hepatitis C The virus tends to cause chronic infection in 70%–80% of the infected cases in compared to HBV which induces chronicity in only 10% of infected cases.[66] HCV carcinogenesis is mediated by the viral‐induced factors and host‐induced immunologic response.”
Identification and Characterization of MAVS, a Mitochondrial Antiviral Signaling Protein that Activates NF-κB and IRF3 (2005, neutral): “The localization of MAVS to the mitochondria provides a strategic position to detect viral replication, which often occurs in intracellular organelles such as ER. For example, the replication of hepatitis C virus (HCV) RNA occurs in a membranous web that connects ER and mitochondria ( Rehermann and ”
Genetic variation in IL28B and spontaneous clearance of hepatitis C virus (2009, neutral): “Subjects in this study were participants in one of six studies: (1) AIDS Link to Intravenous experience (ALIVE(21); n 5 281); (2) Multicentre Hemophilia Cohort Study (MHCS(22); n 5 305); (3) Hemophilia Growth and Development Study (HGDS(23); n 5 106); (4) Correlates of Resolved Versus Low Level Vire”
Mechanism of action of interferon and ribavirin in treatment of hepatitis C (2005, background): “Similarly, pegylated interferon therapy of chronic hepatitis C has been reported to be associated with enhanced T-cell responses in sustained responders in some reports, whereas others have found no induction of T-cell responses regardless of treatment outcom”
Therapeutic potential of Toll-like receptor 9 activation (2006, background): “Spontaneous viral clearance has been associated with early and strong innate immune activation, which generates a strong and diverse T H 1 and CD8 cytolytic T-cell response against the virus, demonstrating the potential for an immune response to control the viru”
Senescence of activated stellate cells limits liver fibrosis (2008, background): “, 2000), while immunostimulatory therapy has a protective effect (Rehermann and Nascimbeni, 2005).”