Neutrophils in the activation and regulation of innate and adaptive immunity
By A. Mantovani, M. Cassatella, C. Costantini, S. Jaillon. Nature reviews. Immunology, 2011
2,925 citations recorded by the source corpus.
Subfields: Immunology
View Paper: https://www.semanticscholar.org/paper/598143ea99fe5d9624413f1e1aba63a0bf9c3480
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In Immunology
- A. Mantovani
- C. Garlanda
- Nikolaos G. Frangogiannis
- F. Balkwill
- R. Ransohoff
- A. Mócsai
- Melissa A Brown
- M. Perretti
Citations in the corpus
- Sexual Dimorphism in Innate Immunity (2019, positive): “Accordingly, primary immunodeficiencies associated with neutropenia or neutrophil dysfunction lead to recurrent infections and life-threatening conditions (see below) [32, 34].”
- Systemic Immune-Inflammation Index Predicts Prognosis of Patients after Curative Resection for Hepatocellular Carcinoma (2014, background): “cates that neutrophils can enhance cancer cell invasion, proliferation, and metastasis as well as assist cancer cells with evading immune surveillance (16, 32).”
- THE INTERLEUKIN-1 FAMILY: BACK TO THE FUTURE (2013, positive): “IL-1 markedly prolongs the lifespan and stimulates the effector function of neutrophils and macrophages (Mantovani et al., 2011).”
- Innate immunity in the central nervous system. (2012, positive): “Once activated, neutrophils upregulate CD15 and CD11b, adhesion molecules that enhance their association with endothelium and migration into tissues (120).”
- The Multifaceted Functions of Neutrophils (2013, background): “Much of this cross talk is mediated by the ability of neutrophils to secrete a host of cytokines or express a large number of cell surface molecules that directly interact with other immune cells (9).”
- Recent advances in 2D and 3D in vitro systems using primary hepatocytes, alternative hepatocyte sources and non-parenchymal liver cells and their use in investigating mechanisms of hepatotoxicity, cell signaling and ADME (2013, positive): “In early studies, their contribution to hepatotoxicity was assessed by treating acetaminophenintoxicated mice with gadolinium chloride (GdCl3), a compound that inhibits phagocytic activity and generation of ROS in macrophages (Lee et al. 2004), and depletes macrophages in the periportal zone of the ”
- Tumor-associated neutrophils: friend or foe? (2012, neutral): “important role in cancer biology [2,3,5-8].”