The endoplasmic reticulum: a multifunctional signaling organelle.
By M. Berridge. Cell Calcium, 2002
967 citations recorded by the source corpus.
Subfields: Cell Biology, Epidemiology
View Paper: https://www.semanticscholar.org/paper/3d22fad6cdac382ca137e7c31742daac56e149d4
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Cell Biology
- J. Malhotra
- A. Görlach
- Peter Klappa
- Dr. Thomas Kietzmann
- R. Kaufman
- M. Cnop
- Decio L. Eizirik
- A. K. Cardozo
In Epidemiology
- J. Malhotra
- A. Görlach
- Peter Klappa
- R. Kaufman
- Dr. Thomas Kietzmann
- Alexei Verkhratsky
- M. Cnop
- Decio L. Eizirik
Citations in the corpus
- The Endoplasmic Reticulum: Folding, Calcium Homeostasis, Signaling, and Redox Control (2006, positive): “store participates in the generation of rapid intracellular Ca 2 signals following chemical activation of plasmalemmal receptors or electrical excitation of the plasma membrane (14 2+ buffer, which is required to remove excess Ca 2+ accumulated during cellular stimulation (246).”
- Mutation of junctophilin type 2 associated with hypertrophic cardiomyopathy (2007, background): “Functional communications between cell-surface and intracellular channels are essential features of excitable cells (Berridge 2002).”
- Store-operated calcium channels. (2005, neutral): “The ER is a multifarious organelle carrying out a litany of interdependent processes (26).”
- Endoplasmic Reticulum Stress and Oxidative Stress: A Vicious Cycle or a Double-Edged Sword? (2007, positive): “Furthermore, ROS can also feedback to sensitize the Ca 2 (cid:2) -release channels at the ER membrane (5, 146).”
- Physiology and pathophysiology of the calcium store in the endoplasmic reticulum of neurons. (2005, background): “Therefore, the ER may be defined as a multifunctional organelle able to detect and integrate incoming signals and generate output signals in response to environmental changes (40, 57).”
- The role for endoplasmic reticulum stress in diabetes mellitus. (2008, background): “This gradient is generated by the sarco(endo)plasmic reticulum Ca(2) ATPase (SERCA) proteins, which pump Ca(2) into the ER, and the Ins(1,4,5)P3 and ryanodine receptors that release Ca(2) from the ER (29).”