Physiology and pathophysiology of the calcium store in the endoplasmic reticulum of neurons.

By Alexei Verkhratsky. Physiological Reviews, 2005

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Physiology of Astroglia. (2018, neutral): “The first two types of intracellular channels dwell in the endomembrane of the endoplasmic reticulum and mediate Ca(2) mobilization from the latter (1805), whereas TPCs are localized to the acidic Ca(2) stores (278).”
Basis for a neuronal version of Grover's quantum algorithm (2014, background): “The fraction of unbound luminal Ca 2 + ranges from 100 to 700 μ M (Montero et al., 1995; Bygrave and Benedetti, 1996; Pinton et al., 1998; Alvarez and Montero, 2002; Bassik et al., 2004; Palmer et al., 2004; Verkhratsky, 2005).”
The Endoplasmic Reticulum: Folding, Calcium Homeostasis, Signaling, and Redox Control (2006, positive): “store participates in the generation of rapid intracellular Ca 2 signals following chemical activation of plasmalemmal receptors or electrical excitation of the plasma membrane (14 2+ buffer, which is required to remove excess Ca 2+ accumulated during cellular stimulation (246).”
ER stress and neurodegenerative diseases (2006, methodology): “Pelizaeus-Merzbacher disease (PMD) is a X-linked recessive pediatric disorder characterized by diffuse hypomyelina- tion of the central nervous system leading to a variety of symptoms.64 Mutations, duplications or deletions in the phopholipid protein (PLP) gene cause PMD with the accumulation of PLP”