Energy intake is associated with endotoxemia in apparently healthy men.

By J. Amar, R. Burcelin, J. Ruidavets, Patrice D Cani, J. Fauvel, M. Alessi, B. Chamontin, J. Ferrières. American Journal of Clinical Nutrition, 2008

600 citations recorded by the source corpus.

Subfields: Epidemiology, Molecular Biology, Public Health, Environmental and Occupational Health

View Paper: https://www.semanticscholar.org/paper/3be3c14a3365720c415a0bcad195128add7c6171

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Epidemiology

1. Herbert Tilg
2. Patrice D Cani
3. A. Moschen
4. N. Delzenne

In Molecular Biology

1. Patrice D Cani
2. Herbert Tilg
3. N. Delzenne
4. Rosa Krajmalnik-Brown
5. J. Furet
6. R. Gambino
7. G. Musso
8. G. Corthier

Citations in the corpus

• INFLAMMATION AND LIPID SIGNALING IN THE ETIOLOGY OF INSULIN RESISTANCE (2012, unknown): “In addition to the potential roles of SFAs as endogenous activators of Tlr signaling, low levels of endotoxin derived from bowel flora are present in the portal circulation, with levels varying dependent on various factors, including diet (Amar et al., 2008; Bäckhed et al., 2004).”
• Interactions between gut microbiota and host metabolism predisposing to obesity and diabetes. (2011, background): “Other observational human studies linked the intake of a high-fat diet to plasma endotoxinemia (79).”
• Intestinal mucosal adherence and translocation of commensal bacteria at the early onset of type 2 diabetes: molecular mechanisms and probiotic treatment (2011, unknown): “Hence, we described this increase in plasma LPS as ’metabolic endotoxemia’.”
• Propensity to high-fat diet-induced obesity in rats is associated with changes in the gut microbiota and gut inflammation. (2010, background): “Indeed, in humans, energy intake has been shown to be associated with LPS plasma levels (2).”
• Human gut microbiome: hopes, threats and promises (2018, background): “22 Indeed, genetic models of both diet-induced obesity and diabetes were characterised by an increased level of circulating LPS, a condition called metabolic endotoxaemia, 22 which was later confirmed in humans (figure 2) 25–34 .”
• Differential Adaptation of Human Gut Microbiota to Bariatric Surgery–Induced Weight Loss (2010, background): “In humans, increased endotoxemia (circulating lipopolysaccharides) was found to be associated with increased fat consumption (10).”
• The role of the gut microbiota in energy metabolism and metabolic disease. (2009, background): “Along the same line, in a large sample of men (n=211) from a population based-study, we found a link between energy (food) intake and metabolic endotoxemia [55].”
• Negative Effects of a High-Fat Diet on Intestinal Permeability: A Review. (2019, background): “This mechanism also confirms findings that consuming diets high in calorie content (fats) is associated with elevated serum LPS in healthy men (93).”