CCAAT/enhancer-binding proteins: structure, function and regulation.

By D. Ramji, P. Foka. Biochemical Journal, 2002

1,445 citations recorded by the source corpus.

Subfields: Immunology, Molecular Biology

View Paper: https://www.semanticscholar.org/paper/2a06d9a899e8c51b9f91d308051ea02323defea9

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Immunology

1. A. Garg
2. Nilesh Amatya
3. S. Gaffen

In Molecular Biology

1. R. Jaenisch
2. J. Hanna
3. C. Lengner
4. Marius Wernig
5. Ruth K. Foreman
6. M. Creyghton
7. S. Markoulaki
8. C. Beard

Citations in the corpus

• cDNA microarray analysis reveals the capacity of the cold-adapted Antarctic fish Trematomus bernacchii to alter gene expression in response to heat stress (2009, background): “For instance, two heat-induced genes encoded the transcriptional regulator CCAAT/enhancer binding protein beta (C/EBP- ), an established antiproliferative agent (Ramji and Foka 2002) and another promoter of cell cycle arrest, the signaling molecule p21-RAC3.”
• Regulation of superoxide dismutase genes: implications in disease. (2009, background): “The binding of C/EBPs to DNA requires that homo- or heterodimerization take place within the various members of the C/EBP family or with other transcription factors [90].”
• The aryl hydrocarbon receptor complex and the control of gene expression. (2008, background): “In particular, C/EBPβ and C/EBPδ are dramatically upregulated by IL-6 or TNF-α and appear to play an important role in sustaining the transcriptional response to inflammation.(319,320) An excellent example of combinatorial transcriptional regulation of inflammatory signaling can be observed on the e”
• Transcription Factors in Long-Term Memory and Synaptic Plasticity (2009, neutral): “The regulatory and functional roles of C/EBPs in various tissues have been reviewed previously (131, 154, 201, 210, 214).”
• Oncogene-induced senescence relayed by an interleukin-dependent inflammatory network. (2008, background): “Indeed, several C/EBPb isoforms (including LIP and LAP, which are generated by differential start site usage and proteolytic cleavage [ Ramji and Foka, 2002 ]), were upregulated during OIS (Figure 4B).”
• IL-17 Signaling: The Yin and the Yang (2017, background): “The biological significance of IL-17-induced changes in alternative translation is unclear, but one of the alternatively generated C/EBPβ isoforms contains only the DNA binding domain and is thus potentially a transcriptional repressor [71, 84].”
• Structure and signalling in the IL-17 receptor superfamily (2009, neutral): “C/EBPβ exists in three isoforms that are generated by alternative translatio”
• Direct reprogramming of terminally differentiated mature B lymphocytes to pluripotency. (2008, positive): “This was obtained either by overexpressing C/EBPa transcription factor, which normally plays a role in granulocyte cell fate specification (Ramji and Foka, 2002), or by knockdown of Pax5, a key transcription factor in B cells.”