Psychomotor Stimulant Addiction: A Neural Systems Perspective

By Barry J. Everitt, M. Wolf. Journal of Neuroscience, 2002

703 citations recorded by the source corpus.

Subfields: Cellular and Molecular Neuroscience, Cognitive Neuroscience

View Paper: https://www.semanticscholar.org/paper/17206f287e07fb0be65a56aa5ba2315fb7819bd2

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In Cognitive Neuroscience

Citations in the corpus

Cue-induced activation of the striatum and medial prefrontal cortex is associated with subsequent relapse in abstinent alcoholics (2004, background): “The dorsal and ventral striatum may play a predominant role in the acquisition and maintenance of cue-induced drug and alcohol intake (Di Chiara 2002; Everitt and Wolf 2002 ).”
Cocaine seeking habits depend upon dopamine-dependent serial connectivity linking the ventral with the dorsal striatum. (2008, positive): “Such compulsive behavior, defined as persisting despite negative consequences (see also the Diagnostic and Statistical Manual IV; American Psychiatric Association, 1994) emerges only after long self-administration sessions and an extended, but not a brief, drug-taking history (Deroche-Gamonet et al.”
Neuroadaptations in cystine-glutamate exchange underlie cocaine relapse (2003, background): “jpg" NDATA ITEM> ]> Addiction to cocaine is marked by a transition from recreational drug use to a compulsive pattern in which addiction-related behaviors such as craving and paranoia emerg”
Adult neurogenesis: from precursors to network and physiology. (2005, background): “These results suggest that memory traces of the association between a specific context and the availability of the drug could be stored in the HF, and the hypothesis that drug addiction is an aberrant form of learning mediated by maladaptive recruitments of hippocampal-dependent memory system has re”
Neurocircuitry of Addiction (2009, background): “In contrast, cue-induced reinstatement appears to involve the basolateral amygdala as a critical substrate with a possible feed-forward mechanism through the prefrontal cortex system involved in drug-induced reinstatement (Everitt and Wolf, 2002; Weiss et al, 2001).”
Synaptic Plasticity: Multiple Forms, Functions, and Mechanisms (2008, background): “Excitatory synaptic transmission in these structures is critical for mediating several forms of longlasting, drug-induced behavioral plasticity (Everitt and Wolf, 2002; Hyman and Malenka, 2001).”
Is there a common molecular pathway for addiction? (2005, background): “Consistent with common mechanisms of addiction are the observations that certain drugs of abuse, under particular experimental conditions, can induce cross-tolerance and cross-sensitization to one another with respect to their locomotor activating and rewarding effect”
Addiction and the brain antireward system. (2008, background): “…projection from the basolateral amygdala to the nucleus accumbens as a critical substrate with a possible feed-forward mechanism through the prefrontal cortex system involved in drug-induced reinstatement and dopamine modulation in the basolateral amygdala (Everitt & Wolf 2002, Weiss et al. 2001).”
Neural mechanisms of addiction: the role of reward-related learning and memory. (2006, background): “…mechanisms similar to those underlying all other forms of associative longterm memory (Di Chiara 1998, Berke & Hyman 2000, Hyman & Malenka 2001, Everitt & Wolf 2002, Robbins & Everitt 2002, Chao & Nestler 2004, Hyman 2005), namely by complex and bidirectional activity– dependent changes in the…”
Evidence for sugar addiction: behavioral and neurochemical effects of intermittent, excessive sugar intake. (2008, background): “Extracellular DA increases in the NAc in response to both addictive drugs (De Vries and Shippenberg, 2002; Di Chiara and Imperato, 1988; Everitt and Wolf, 2002; Hernandez and Hoebel, 1988; Hurd et al., 1988; Picciotto and Corrigall, 2002; Pothos et al., 1991) and drug-associated stimuli (Ito et al.”