Calcium and oxidative stress: from cell signaling to cell death.

By G. Ermak, K. Davies. Molecular Immunology, 2002

851 citations recorded by the source corpus.

Subfields: Cell Biology, Molecular Biology

View Paper: https://www.semanticscholar.org/paper/0bcaadbfea11734e77f12b3b8c1e219a8bcf2e91

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In Cell Biology

1. M. Schrader
2. A. Görlach

In Molecular Biology

1. Ramamurthy Mahalingam
2. Nina V. Fedoroff

Citations in the corpus

• Selective Neuronal Vulnerability to Oxidative Stress in the Brain (2010, neutral): “During OS, ROS/RNS usually activate Ca2+ channels and repress Ca2+ pumps (Ermak and Davies, 2002), resulting in elevation of [Ca2+] i and initiation of downstream events mentioned above.”
• Apoptosis, autophagy, and more. (2004, neutral): “…& Lipton, 2003; Brunk & Terman,605 2002; Cho et al., 2003; Cuervo & Dice, 2000; Del606 Roso et al., 2003; Dirks & Leeuwenburgh, 2004;607 Donati et al., 2001; Ermak & Davies, 2002; Fraker608 & Lill-Elghanian, 2004; Nixon, Cataldo, & Mathews,609 2000; Terman et al., 2003; Warner, 2004).”
• Calcium and ROS: A mutual interplay (2015, background): “ROS at moderate levels represent significant signaling molecules, which are widely involved in physiological processes through oxidizing proteins, lipids and polynucleotides [47,75].”
• Oxidative Stress and Neurodegenerative Diseases: A Review of Upstream and Downstream Antioxidant Therapeutic Options (2009, background): “Most significant ill effect on neuronal health takes place by dysregulation of intracellular calcium signaling pathways initiated by ROS and in recent years heavy evidences suggest role of ROS in neuronal cell death [22].”
• Stress response, cell death and signalling: the many faces of reactive oxygen species (2003, background): “But although the concept of redox regulation is an old one in enzymology, there is a new and growing awareness that ROS and redox regulation are central to cell signalling and to both transcriptional and post-transcriptional regulation (Sauer et al. 2001, Droge 2002, Ermak and Davies 2002).”
• Peroxisomes and oxidative stress. (2006, background): “006 heavy metals, ionizing and UV irradiation, heat shock, and inflammation [1].”