Pathogenic Escherichia coli
By J. Kaper, J. Nataro, H. Mobley. Nature Reviews Microbiology, 2004
5,621 citations recorded by the source corpus.
Subfields: Endocrinology, Food Science, Infectious Diseases
View Paper: https://www.semanticscholar.org/paper/0a49c156ca2f54fc9b290e3a3d267b45894b2587
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Endocrinology
In Food Science
- Curtis Huttenhower
- Nicola Segata
- Eric A. Franzosa
- A. V. Van Bruggen
- B. B. Finlay
- E. Franz
- Patrice D Cani
- P. Nordmann
In Infectious Diseases
- G. Peters
- J. Pitout
- B. B. Finlay
- David A. Rasko
- Curtis Huttenhower
- Patrice D Cani
- Nicola Segata
- R. Vasudevan
Citations in the corpus
- Rapid and Easy In Silico Serotyping of Escherichia coli Isolates by Use of Whole-Genome Sequencing Data (2015, background): “24 AY337479 1,842 (1) AY250002* H20 fliC 1 1,731 AY250003 H21 fliC 1 1,476 AIHL01000060 H23 fliC 1 1,767 AB028476* H24 fliC 1 1,482 K72 (H25w)* H25 fliC 1 1,332 AGSG01000116 H26 fliC 2 1,674 99.”
- The Human Gut Microbiome: From Association to Modulation. (2018, neutral): “Indeed, some strains of Escherichia coli are highly beneficial probiotics (Wassenaar, 2016), whereas others are potent pathogens (Kaper et al., 2004).”
- The Pangenome Structure of Escherichia coli: Comparative Genomic Analysis of E. coli Commensal and Pathogenic Isolates (2008, background): “a tropism for the colon, while the latter resides in the small intestine (29).”
- Characterisation of the Escherichia coli strain associated with an outbreak of haemolytic uraemic syndrome in Germany, 2011: a microbiological study. (2011, background): “In addition to harbouring a mixture of virulence loci of two different intestinal E coli pathotypes, all O104:H4 outbreak isolates also express the phenotypes that define Shiga-toxin-producing E coli and enteroaggregative E coli, [15] and [16] specifically production of Shiga-toxin 2 (Vero cell cyto”
- Enterohemorrhagic Escherichia coli O157∶H7 Gene Expression Profiling in Response to Growth in the Presence of Host Epithelia (2009, neutral): “EHEC disease pathogenesis is initiated following pathogen contact with host epithelial cells, and subsequently results in microbial adhesion, toxin production, and modulation of host cell signal transduction cascades [3,4].”
- High-Specificity Targeted Functional Profiling in Microbial Communities with ShortBRED (2015, background): “For example, Escherichia and Salmonella are closely related bacterial genera that contain many human pathogens [25, 26]; both were highly enriched for AR determinants.”
- Antimicrobial Resistance in Escherichia coli (2018, background): “coli isolates can be classified into different pathotypes, or pathovars, where each pathotype causes a different disease (1).”
- Recent Advances in Understanding Enteric Pathogenic Escherichia coli (2013, background): “LEE-negative STEC strains have also been isolated in cases of HC and HUS, but since they lack the T3SS, colonization and pathogenesis are likely caused by a unique set of virulence factors.”
- Human Intestinal Barrier Function in Health and Disease (2016, background): “coli also possess an attaching and effacement locus, but exert less profound effects on the barrier.(16) One reported difference is the increased expression of claudin-2, which forms cationselective channels in the paracellular space, resulting in water transportation across the TJs.”
- The Pangenome: Diversity, Dynamics and Evolution of Genomes (2020, background): “E. coli strains are grouped into pathovars based on the presence of virulence markers, often encoded on mobile elements (Kaper et al. 2004).”