Receptor interacting protein kinase-3 determines cellular necrotic response to TNF-alpha.
By Sudan He, Lai Wang, Lin Miao, Tao Wang, Fenghe Du, Liping Zhao, Xiaodong Wang. Cell, 2009
1,879 citations recorded by the source corpus.
Subfields: Cancer Research, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/05cdfcf96e3786f9db280d17e8d35845258955b4
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In Cancer Research
In Molecular Biology
- T. Kanneganti
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- A. Strasser
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- R. Malireddi
Citations in the corpus
- Mixed lineage kinase domain-like protein MLKL causes necrotic membrane disruption upon phosphorylation by RIP3. (2014, background): “The shape of the phospho-MLKL signal in the dying cells reminded us of the formation of RIP3 puncta during necroptosis (He et al., 2009; Sun et al., 2012).”
- ZBP1 and TAK1: Master Regulators of NLRP3 Inflammasome/Pyroptosis, Apoptosis, and Necroptosis (PAN-optosis) (2019, background): “…and activation of the protein mixed lineage kinase domain-like pseudokinase (MLKL) that forms pores in the membrane to execute necroptosis (Feng et al., 2007; Degterev et al., 2008; Cho et al., 2009; He et al., 2009; Zhang et al., 2009; Sun et al., 2012; Zhao et al., 2012; Green, 2019).”
- Molecular definitions of cell death subroutines: recommendations of the Nomenclature Committee on Cell Death 2012 (2011, neutral): “citotoxins and the ligation of death receptors, at least under selected circumstances.(9,11,12,84,85) Indeed, when caspases (and in particular caspase-8) are inhibited by genetic”
- Necroptosis: a regulated inflammatory mode of cell death (2018, background): “Microglial-mediated chronic inflammation further raises questions how programmed necrosis regulates the chronic inflammation after SCI. Multiple sclerosis is an autoimmune disease of the brain characterized by demyelination and chronic inflammation.”
- Cellular Stress Responses: Cell Survival and Cell Death (2010, background): “To this end, RIP3 was identified in an RNA interference screen to be essential for necrosis in response to TNFα stimulation and during virus infection [72, 73].”
- Cell Biology of Ischemia/Reperfusion Injury (2012, neutral): “However, RIP1 and RIP3 have also been shown to be critical for the progression of necrotic death in a variety of cell lines (Cho et al., 2009; Degterev et al., 2005; He et al., 2009; Holler et al., 2000).”