Molecular definitions of cell death subroutines: recommendations of the Nomenclature Committee on Cell Death 2012
By L. Galluzzi, Ilio Vitale, J. Abrams, E. Alnemri, Eric H. Baehrecke, M. V. Blagosklonny, T. Dawson, Valina L. Dawson, W. El-Deiry, Simone Fulda and 23 more. Cell Death and Differentiation, 2011
2,468 citations recorded by the source corpus.
Subfields: Epidemiology, Immunology, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/f733f38dada8dfb30a2457b051ea85f613c61483
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In Epidemiology
- Beth Levine
- Guido Kroemer
- D. Klionsky
- N. Mizushima
- I. Slaninová
- Douglas R. Green
- V. Deretic
- Alec C. Kimmelman
In Immunology
In Molecular Biology
- Guido Kroemer
- Beth Levine
- Douglas R. Green
- D. Klionsky
- L. Galluzzi
- I. Slaninová
- N. Mizushima
- Brent R. Stockwell
Citations in the corpus
- Regulation of Ferroptotic Cancer Cell Death by GPX4 (2014, background): “Cells can undergo regulated forms of cell death in a variety of contexts (Galluzzi et al., 2012), including during development (Penaloza et al., 2006).”
- Lipotoxicity and the gut-liver axis in NASH pathogenesis. (2018, background): “Necrosis and necroptosis are recently-discovered types of cell death, the contribution of which to NASH is still uncertain [33].”
- Regulation of tumour necrosis factor signalling: live or let die (2015, background): “Unlike the contained self-destruction of an apoptotic cell, a necroptotic cell displays swelling of cellular organelles, rupture of the cell membrane and uncontrolled release of cellular contents into the surrounding tissue, followed ultimately by cell death 92,93 .”
- Consensus guidelines for the detection of immunogenic cell death (2014, background): “Thus, although apoptosis as a physiological process involved in (post-) embryonic development and tissue homeostasis invariably fails to engage the adaptive branch of the immune system,(24,25) specific stimuli can promote an immunogenic variant of regulated cell death that manifests with both morpho”
- Guidelines for the use and interpretation of assays for monitoring autophagy (2012, background): “Clearly claim (b) is stronger than claim (a), and needs to be justified by proof that inhibiting autophagy, through either genetic or chemical means, prevents cell death.(669) Claim (c) is still stronger, because, even if the cell death is blocked by autophagy inhibition, proof needs to be provided ”
- Essential versus accessory aspects of cell death: recommendations of the NCCD 2015 (2014, methodology): “Apparently at odds with the role of PARP1 in the execution of parthanatos, both the Parp1 / genotype and the administration of (relatively unselective) PARP inhibitors have been associated with bona fide cytoprotection in rodent models of ischemia/reperfusion injury and retinal degeneration.318–321 ”