Myogenic factors that regulate expression of muscle-specific microRNAs.

By Prakash K. Rao, Roshan M. Kumar, Mina Farkhondeh, S. Baskerville, H. Lodish. Proceedings of the National Academy of Sciences of the United States of America, 2006

732 citations recorded by the source corpus.

Subfields: Cancer Research, Molecular Biology

View Paper: https://www.semanticscholar.org/paper/017b82aa492624ec2d5e43d661ebb4e17665fce4

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Citations in the corpus

microRNA-1 and microRNA-206 regulate skeletal muscle satellite cell proliferation and differentiation by repressing Pax7 (2010, background): “Activated MyoD in satellite cell–derived myogenic progenitor cells will then turn on miR-1 and miR-206 (Rao et al., 2006; Rosenberg et al., 2006).”
MicroRNA-133 controls cardiac hypertrophy (2007, background): “In mouse skeletal muscle, MyoD and myogenin modulate the transcription of miR-1 and miR-133 according to a regulatory loo”
Transactivation of miR-34a by p53 broadly influences gene expression and promotes apoptosis. (2007, background): “For instance, a number of well-studied mammalian transcription factors including c-Myc, cAMP-response element binding protein (CREB), and MyoD have been shown to control both mRNA and miRNA expression (O’Donnell et al., 2005;Rao et al., 2006;Vo et al., 2005;Zhao et al., 2005).”
MicroRNA regulation of cell lineages in mouse and human embryonic stem cells. (2008, unknown): “…regulators of muscle differentiation: serum response factor (SRF), MyoD, and Mef2 (Zhao et al., 2005; Kwon et al., 2005; Sokol and Ambros, 2005; Rao et al., 2006). miR-1 promotes differentiation of cardiac progenitors and exit from the cell cycle in mammals and in flies (Zhao et al., 2005,…”
MEF2: a central regulator of diverse developmental programs (2007, background): “Recently, several microRNAs were identified that affect skeletal muscle differentiation and proliferation (Boutz et al., 2007; Chen et al., 2006; Kim et al., 2006; Rao et al., 2006).”
Dysregulation of cardiogenesis, cardiac conduction, and cell cycle in mice lacking miRNA-1-2. (2007, background): “…-133 are expressed in cardiac and skeletal muscle and are transcriptionally regulated by the myogenic differentiation factors MyoD, Mef2, and serum response factor (SRF) (Chen et al., 2006; Kwon et al., 2005; Lagos-Quintana et al., 2001; Rao et al., 2006; Sokol and Ambros, 2005; Zhao et al., 2005).”
A Long Noncoding RNA Controls Muscle Differentiation by Functioning as a Competing Endogenous RNA (2011, unknown): “This region contains E-box sequences (CANNTG) previously shown to be functional for MyoD association (Rao et al., 2006) and mir-206 expression (Williams et al.”
The muscle-specific microRNA miR-1 regulates cardiac arrhythmogenic potential by targeting GJA1 and KCNJ2 (2011, background): “Of more than 300 miRNAs that have been identified, miR-1 and miR-133 are considered to be muscle specifi”
MicroRNAs in development and disease. (2011, background): “Examples include the mouse miR-1–1 and miR-1–2 promoters that are regulated by serum response factor (SRF), myoD, and muscle enhancer factor 2 (Mef2c), also thought to cotranscribe miR-133a-2 and miR-133a-1, respectively, as part of bicistronic transcripts (713), and the miR-206 promoter, which bind”