É. W. Khandjian
Trends in Genetics · 2008
A researcher in the Science corpus: 1 paper; trusts 0 researchers and is trusted by 0 researchers here, as inferred from the citation graph.
Papers from É. W. Khandjian's perspective
Ordered by how strongly the researchers É. W. Khandjian trusts endorse each paper. Same corpus, a different order for every researcher.
- Mechanisms of BCR–ABL in the pathogenesis of chronic myelogenous leukaemia (Nature Reviews. Cancer)
- Hyperactive Ras in developmental disorders and cancer (Nature Reviews. Cancer)
- Targeting RAS signalling pathways in cancer therapy (Nature Reviews. Cancer)
- Mutations in PTPN11, encoding the protein tyrosine phosphatase SHP-2, cause Noonan syndrome (Nature Genetics)
- The RAF proteins take centre stage (Nature reviews. Molecular cell biology)
- Activated Kras and Ink4a/Arf deficiency cooperate to produce metastatic pancreatic ductal adenocarcinoma. (Genes & Development)
- Tumor induction by an endogenous K-ras oncogene is highly dependent on cellular context. (Cancer Cell)
- Somatic activation of the K-ras oncogene causes early onset lung cancer in mice (Nature)
- Mechanism of activation of the RAF-ERK signaling pathway by oncogenic mutations of B-RAF.
- High frequency of BRAF mutations in nevi (Nature Genetics)
Their papers
- RNA-binding proteins in human genetic disease. (2008) — 820 citations