Mechanisms of BCR–ABL in the pathogenesis of chronic myelogenous leukaemia

By R. Ren. Nature Reviews. Cancer, 2005

1,054 citations recorded by the source corpus.

Subfields: Hematology, Immunology, Rheumatology

View Paper: https://www.semanticscholar.org/paper/4ff35e6eb954811d84486db9f08d5a018f29e171

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Hematology

1. Trey Ideker
2. S. Richard
3. Janusz Dutkowski
4. Kai-Wei Chang
5. Kiven Erique Lukong
6. É. W. Khandjian

In Immunology

1. Trey Ideker
2. S. Richard
3. Janusz Dutkowski
4. Kai-Wei Chang
5. Kiven Erique Lukong
6. É. W. Khandjian

In Rheumatology

1. Trey Ideker
2. S. Richard
3. Janusz Dutkowski
4. Kai-Wei Chang
5. Kiven Erique Lukong
6. É. W. Khandjian

Citations in the corpus

• Protein Networks as Logic Functions in Development and Cancer (2011, background): “Protein combinations are equally instrumental in the pathogenesis of human disease, for instance the inappropriate fusion of Bcr and Abl that leads to chronic myelogenous leukemia [4] or the abnormal interactions acquired by the huntington protein in Huntington’s Disease [5].”
• RNA-binding proteins in human genetic disease. (2008, positive): “Chromosomal alterations such as the Philadelphia translocation, generated by a reciprocal translocation between chromosomes 9 and 22, results in chronic myeloid leukemia [ 66 ].”