Nina E. Shepardson

Physiology · 2008–2011

A researcher in the Science corpus: 4 papers; trusts 3 researchers and is trusted by 2 researchers here, as inferred from the citation graph.

Subfields

• Physiology — 39 citations into it; #1,392 of 2,276 by trust here, #537 by citations
• Cellular and Molecular Neuroscience — 30 citations into it; #2,156 of 2,998 by trust here, #549 by citations

Whom Nina E. Shepardson trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

1. G. Shankar
2. Matthew Townsend

Who trusts Nina E. Shepardson overall

1. D. Selkoe
2. L. Mucke

Papers from Nina E. Shepardson's perspective

Ordered by how strongly the researchers Nina E. Shepardson trusts endorse each paper. Same corpus, a different order for every researcher.

1. Structural basis of long-term potentiation in single dendritic spines (Nature)
2. Activation of synaptic NMDA receptors induces membrane insertion of new AMPA receptors and LTP in cultured hippocampal neurons. (Neuron)
3. Regulation of NMDA receptor trafficking by amyloid-β (Nature Neuroscience)
4. Role of NMDA Receptor Subtypes in Governing the Direction of Hippocampal Synaptic Plasticity (Science)
5. Enhanced Neurofibrillary Degeneration in Transgenic Mice Expressing Mutant Tau and APP (Science)
6. LTP inhibits LTD in the hippocampus via regulation of GSK3beta. (Neuron)
7. Extrasynaptic NMDARs oppose synaptic NMDARs by triggering CREB shut-off and cell death pathways (Nature Neuroscience)
8. Aberrant excitatory neuronal activity and compensatory remodeling of inhibitory hippocampal circuits in mouse models of Alzheimer's disease. (Neuron)
9. Effects of secreted oligomers of amyloid β‐protein on hippocampal synaptic plasticity: a potent role for trimers (Journal of Physiology)
10. Differential Roles of NR2A and NR2B-Containing NMDA Receptors in Cortical Long-Term Potentiation and Long-Term Depression (Journal of Neuroscience)

Their papers

• Soluble amyloid β-protein dimers isolated from Alzheimer cortex directly induce Tau hyperphosphorylation and neuritic degeneration (2011) — 926 citations
• Soluble Aβ oligomers inhibit long-term potentiation through a mechanism involving excessive activation of extrasynaptic NR2B-containing NMDA receptors (2011) — 644 citations
• Soluble oligomers of amyloid β-protein facilitate hippocampal long-term depression by disrupting neuronal glutamate uptake (2009) — 909 citations
• Amyloid-β protein dimers isolated directly from Alzheimer's brains impair synaptic plasticity and memory (2008) — 2,572 citations