H. Yee

Cell · with Dickins · 2008

A researcher in the Science corpus: 1 paper; trusts 1 researcher and is trusted by 2 researchers here, as inferred from the citation graph.

Whom H. Yee trusts

Their strongest outbound trust, strongest first, inferred from their citations.

  1. M. Pinzani (in Hepatology)

Who trusts H. Yee

  1. J. Campisi (in Physiology)
  2. S. Friedman (in Immunology)

Papers from H. Yee's perspective

Ordered by how strongly the researchers H. Yee trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Liver fibrosis (Springer Seminars in Immunopathology)
  2. Rb-mediated heterochromatin formation and silencing of E2F target genes during cellular senescence. (Cell)
  3. Tumour biology: Senescence in premalignant tumours (Nature)
  4. A senescence program controlled by p53 and p16INK4a contributes to the outcome of cancer therapy. (Cell)
  5. The DNA damage pathway regulates innate immune system ligands of the NKG2D receptor (Nature)
  6. Oncogene-induced senescence is a DNA damage response triggered by DNA hyper-replication (Nature)
  7. Natural killer cells ameliorate liver fibrosis by killing activated stellate cells in NKG2D-dependent and tumor necrosis factor-related apoptosis-inducing ligand-dependent manners. (Gastroenterology)
  8. Senescent fibroblasts promote epithelial cell growth and tumorigenesis: A link between cancer and aging (Proceedings of the National Academy of Sciences of the United States of America)
  9. Senescence and tumour clearance is triggered by p53 restoration in murine liver carcinomas (Nature)
  10. Oncogene-induced senescence is part of the tumorigenesis barrier imposed by DNA damage checkpoints (Nature)

Their papers