W. Cawthorn
Epidemiology · 2007
A researcher in the Science corpus: 1 paper; trusts 4 researchers and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Epidemiology — 22 citations into it
- Physiology — 18 citations into it
- Molecular Biology — 10 citations into it
- Cancer Research — 6 citations into it
Whom W. Cawthorn trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Papers from W. Cawthorn's perspective
Ordered by how strongly the researchers W. Cawthorn trusts endorse each paper. Same corpus, a different order for every researcher.
- Chronic inflammation in fat plays a crucial role in the development of obesity-related insulin resistance. (Journal of Clinical Investigation)
- Obesity is associated with macrophage accumulation in adipose tissue. (Journal of Clinical Investigation)
- Disruption of Adiponectin Causes Insulin Resistance and Neointimal Formation* (Journal of Biological Chemistry)
- Adipocyte death defines macrophage localization and function in adipose tissue of obese mice and humans Published, JLR Papers in Press, September 8, 2005. DOI 10.1194/jlr.M500294-JLR200 (Journal of Lipid Research)
- A central role for JNK in obesity and insulin resistance (Nature)
- Obesity induces a phenotypic switch in adipose tissue macrophage polarization. (Journal of Clinical Investigation)
- IKK-beta links inflammation to obesity-induced insulin resistance. (Nature Medicine)
- Reactive oxygen species have a causal role in multiple forms of insulin resistance (Nature)
- Adiponectin stimulates glucose utilization and fatty-acid oxidation by activating AMP-activated protein kinase (Nature Medicine)
- The hormone resistin links obesity to diabetes (Nature)
Their papers
- TNF-α and adipocyte biology (2007) — 728 citations