C. Steenbergen
Molecular Biology · 2008
A researcher in the Science corpus: 1 paper; trusts 4 researchers and is trusted by 1 researcher here, as inferred from the citation graph.
Subfields
- Molecular Biology — 27 citations into it; #15,872 of 17,773 by trust here, #14,176 by citations
- Pathology and Forensic Medicine — 10 citations into it; #335 of 414 by trust here, #298 by citations
Whom C. Steenbergen trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts C. Steenbergen overall
Papers from C. Steenbergen's perspective
Ordered by how strongly the researchers C. Steenbergen trusts endorse each paper. Same corpus, a different order for every researcher.
- The ADP/ATP translocator is not essential for the mitochondrial permeability transition pore (Nature)
- Cyclophilin D-dependent mitochondrial permeability transition regulates some necrotic but not apoptotic cell death (Nature)
- Preconditioning the myocardium: from cellular physiology to clinical cardiology. (Physiological Reviews)
- Inhibition of myocardial injury by ischemic postconditioning during reperfusion: comparison with ischemic preconditioning. (American Journal of Physiology. Heart and Circulatory Physiology)
- Growing roles for the mTOR pathway. (Current Opinion in Cell Biology)
- Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death (Nature)
- Autophagic programmed cell death by selective catalase degradation. (Proceedings of the National Academy of Sciences of the United States of America)
- SIN1/MIP1 maintains rictor-mTOR complex integrity and regulates Akt phosphorylation and substrate specificity. (Cell)
- Prolonged rapamycin treatment inhibits mTORC2 assembly and Akt/PKB. (Molecules and Cells)
- The mitochondrial permeability transition from in vitro artifact to disease target (The FEBS Journal)
Their papers
- Mechanisms underlying acute protection from cardiac ischemia-reperfusion injury. (2008) — 1,498 citations