A. Colige
Cancer Research · 1995–2017
A researcher in the Science corpus: 7 papers; trusts 1 researcher and is trusted by 3 researchers here, as inferred from the citation graph.
Subfields
- Cancer Research — 7 citations into it; #1,304 of 1,780 by trust here, #1,399 by citations
- Hematology — 7 citations into it
- Immunology and Allergy — 5 citations into it; #103 of 206 by trust here, #91 by citations
Whom A. Colige trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts A. Colige overall
Papers from A. Colige's perspective
Ordered by how strongly the researchers A. Colige trusts endorse each paper. Same corpus, a different order for every researcher.
- METH-1, a Human Ortholog of ADAMTS-1, and METH-2 Are Members of a New Family of Proteins with Angio-inhibitory Activity*
- cDNA cloning and expression of bovine procollagen I N-proteinase: a new member of the superfamily of zinc-metalloproteinases with binding sites for cells and other matrix components.
- Association of a nsSNP in ADAMTS14 to some osteoarthritis phenotypes.
- The ADAMTS metalloproteinases.
- The hallmarks of cancer (RNA Biology)
- Domains and Maturation Processes That Regulate the Activity of ADAMTS-2, a Metalloproteinase Cleaving the Aminopropeptide of Fibrillar Procollagens Types I–III and V*
- CD36: a class B scavenger receptor involved in angiogenesis, atherosclerosis, inflammation, and lipid metabolism. (Journal of Clinical Investigation)
- Molecular Cloning of a Gene Encoding a New Type of Metalloproteinase-disintegrin Family Protein with Thrombospondin Motifs as an Inflammation Associated Gene*
- TIMP-3 inhibits the procollagen N-proteinase ADAMTS-2.
- Efficient activation of the lymphangiogenic growth factor VEGF-C requires the C-terminal domain of VEGF-C and the N-terminal domain of CCBE1
Their papers
- ADAMTS-2 functions as anti-angiogenic and anti-tumoral molecule independently of its catalytic activity (2010) — 77 citations
- ADAMTS3 activity is mandatory for embryonic lymphangiogenesis and regulates placental angiogenesis (2015) — 92 citations
- Loss of ADAMTS3 activity causes Hennekam lymphangiectasia–lymphedema syndrome 3 (2017) — 105 citations
- The procollagen N-proteinases ADAMTS2, 3 and 14 in pathophysiology. (2015) — 133 citations
- Domains and Maturation Processes That Regulate the Activity of ADAMTS-2, a Metalloproteinase Cleaving the Aminopropeptide of Fibrillar Procollagens Types I–III and V* (2005) — 115 citations
- Procollagen II Amino Propeptide Processing by ADAMTS-3 (2001) — 230 citations
- Characterization and Partial Amino Acid Sequencing of a 107-kDa Procollagen I N-Proteinase Purified by Affinity Chromatography on Immobilized Type XIV Collagen (*) (1995)