G. Iliakis

Molecular Biology · 2006–2008

A researcher in the Science corpus: 2 papers; trusts 2 researchers and is trusted by 0 researchers here, as inferred from the citation graph.

Subfields

Whom G. Iliakis trusts

Their strongest outbound trust, strongest first, inferred from their citations.

  1. Stephen P. Jackson (in Molecular Biology)
  2. M. Löbrich (in Molecular Biology)

Papers from G. Iliakis's perspective

Ordered by how strongly the researchers G. Iliakis trusts endorse each paper. Same corpus, a different order for every researcher.

  1. A critical role for histone H2AX in recruitment of repair factors to nuclear foci after DNA damage. (Current Biology)
  2. Genomic Instability in Mice Lacking Histone H2AX (Science)
  3. MDC1 is a mediator of the mammalian DNA damage checkpoint (Nature)
  4. Histone H2AX phosphorylation is dispensable for the initial recognition of DNA breaks (Nature Cell Biology)
  5. DNA damage activates ATM through intermolecular autophosphorylation and dimer dissociation (Nature)
  6. DNA damage-induced G2–M checkpoint activation by histone H2AX and 53BP1 (Nature Cell Biology)
  7. Methylated lysine 79 of histone H3 targets 53BP1 to DNA double-strand breaks (Nature)
  8. MDC1 directly binds phosphorylated histone H2AX to regulate cellular responses to DNA double-strand breaks. (Cell)
  9. Recombinational DNA double-strand breaks in mice precede synapsis (Nature Genetics)
  10. P53 Binding Protein 1 (53bp1) Is an Early Participant in the Cellular Response to DNA Double-Strand Breaks (Journal of Cell Biology)

Their papers