M. Block
Neurology · Nature Reviews Neuroscience · 2004–2010
A researcher in the Science corpus: 7 papers; trusts 11 researchers and is trusted by 10 researchers here, as inferred from the citation graph.
Subfields
- Neurology — 87 citations into it; #238 of 1,455 by trust here, #137 by citations
- Immunology — 38 citations into it; #844 of 2,160 by trust here, #1,050 by citations
- Cellular and Molecular Neuroscience — 34 citations into it; #2,685 of 2,998 by trust here, #1,161 by citations
- Physiology — 29 citations into it; #658 of 2,276 by trust here, #942 by citations
- Molecular Biology — 12 citations into it
Whom M. Block trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts M. Block overall
- Melinda E Lull
- M. Tansey
- P. Whitton
- Wei-Wei Chen
- David J. Loane
- Yuanyuan Tang
- S. Lehnardt
- Rafael Franco
- U. Hanisch
- J. Felger
Papers from M. Block's perspective
Ordered by how strongly the researchers M. Block trusts endorse each paper. Same corpus, a different order for every researcher.
- Regional Difference in Susceptibility to Lipopolysaccharide-Induced Neurotoxicity in the Rat Brain: Role of Microglia (Journal of Neuroscience)
- NADPH Oxidase Mediates Lipopolysaccharide-induced Neurotoxicity and Proinflammatory Gene Expression in Activated Microglia* (Journal of Biological Chemistry)
- Innate immunity: the missing link in neuroprotection and neurodegeneration? (Nature Reviews Neuroscience)
- Bone marrow-derived microglia play a critical role in restricting senile plaque formation in Alzheimer's disease. (Neuron)
- Activation of innate immunity in the CNS triggers neurodegeneration through a Toll-like receptor 4-dependent pathway (Proceedings of the National Academy of Sciences of the United States of America)
- Aggregated α‐synuclein activates microglia: a process leading to disease progression in Parkinson's disease (The FASEB Journal)
- In-vivo measurement of activated microglia in dementia. (The Lancet)
- The Toll-Like Receptor TLR4 Is Necessary for Lipopolysaccharide-Induced Oligodendrocyte Injury in the CNS (Journal of Neuroscience)
- Ubiquitination of a New Form of α-Synuclein by Parkin from Human Brain: Implications for Parkinson's Disease (Science)
- Altered proteasomal function in sporadic Parkinson's disease. (Experimental Neurology)
Their papers
- Microglia-mediated neurotoxicity: uncovering the molecular mechanisms (2007) — 4,009 citations
- Microglial Activation & Chronic Neurodegeneration (2010) — 982 citations
- Air pollution: mechanisms of neuroinflammation and CNS disease (2009) — 1,460 citations
- Systemic LPS causes chronic neuroinflammation and progressive neurodegeneration (2007) — 2,143 citations
- Microglia and inflammation-mediated neurodegeneration: multiple triggers with a common mechanism. (2005) — 1,572 citations
- Aggregated α‐synuclein activates microglia: a process leading to disease progression in Parkinson's disease (2005) — 1,140 citations
- NADPH Oxidase Mediates Lipopolysaccharide-induced Neurotoxicity and Proinflammatory Gene Expression in Activated Microglia* (2004) — 605 citations