Megan M. Cleland

Molecular Biology · 2006–2010

A researcher in the Science corpus: 3 papers; trusts 2 researchers and is trusted by 4 researchers here, as inferred from the citation graph.

Subfields

Whom Megan M. Cleland trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. Derek P. Narendra
  2. M. Karbowski
  3. R. Youle
  4. R. Phair
  5. D. Suen
  6. Naotada Ishihara

Who trusts Megan M. Cleland overall

  1. I. Slaninová
  2. A. M. van der Bliek
  3. R. Youle
  4. Dieter A. Kubli
  5. D. C. Chan
  6. J. Nunnari
  7. Julia J. Harris

Papers from Megan M. Cleland's perspective

Ordered by how strongly the researchers Megan M. Cleland trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Disruption of Fusion Results in Mitochondrial Heterogeneity and Dysfunction* (Journal of Biological Chemistry)
  2. Mitochondrial dysfunction in Drosophila PINK1 mutants is complemented by parkin (Nature)
  3. OPA1 requires mitofusin 1 to promote mitochondrial fusion. (Proceedings of the National Academy of Sciences of the United States of America)
  4. Parkin is recruited selectively to impaired mitochondria and promotes their autophagy (Journal of Cell Biology)
  5. PINK1/Parkin-mediated mitophagy is dependent on VDAC1 and p62/SQSTM1 (Nature Cell Biology)
  6. PINK1 Is Selectively Stabilized on Impaired Mitochondria to Activate Parkin (PLoS Biology)
  7. The PINK1/Parkin pathway regulates mitochondrial morphology (Proceedings of the National Academy of Sciences of the United States of America)
  8. PINK1-dependent recruitment of Parkin to mitochondria in mitophagy (Proceedings of the National Academy of Sciences of the United States of America)
  9. Loss of PINK1 Function Promotes Mitophagy through Effects on Oxidative Stress and Mitochondrial Fission* (Journal of Biological Chemistry)
  10. Mitochondrial pathology and muscle and dopaminergic neuron degeneration caused by inactivation of Drosophila Pink1 is rescued by Parkin. (Proceedings of the National Academy of Sciences of the United States of America)

Their papers