P. Mehta
Cardiology and Cardiovascular Medicine · with Griendling · 2007
A researcher in the Science corpus: 1 paper; trusts 4 researchers and is trusted by 5 researchers here, as inferred from the citation graph.
Subfields
- Cardiology and Cardiovascular Medicine — 10 citations into it; #316 of 906 by trust here, #639 by citations
- Molecular Biology — 8 citations into it; #8,755 of 17,773 by trust here, #14,833 by citations
- Physiology — 6 citations into it
- Immunology — 5 citations into it
Whom P. Mehta trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts P. Mehta overall
Papers from P. Mehta's perspective
Ordered by how strongly the researchers P. Mehta trusts endorse each paper. Same corpus, a different order for every researcher.
- Angiotensin II Stimulation of NAD(P)H Oxidase Activity: Upstream Mediators (Circulation Research)
- ASK1 is required for sustained activations of JNK/p38 MAP kinases and apoptosis (EMBO Reports)
- ADAMs: key components in EGFR signalling and development
- Signal transduction mechanisms mediating the physiological and pathophysiological actions of angiotensin II in vascular smooth muscle cells. (Pharmacological Reviews)
- NAD(P)H oxidase: role in cardiovascular biology and disease. (Circulation Research)
- Reactive oxygen species in the vasculature: molecular and cellular mechanisms. (HYPERTENSION)
- Mammalian mitogen-activated protein kinase signal transduction pathways activated by stress and inflammation. (Physiological Reviews)
- Protein kinase cascades in the regulation of cardiac hypertrophy. (Journal of Clinical Investigation)
- Novel gp91phox Homologues in Vascular Smooth Muscle Cells: nox1 Mediates Angiotensin II-Induced Superoxide Formation and Redox-Sensitive Signaling Pathways (Circulation Research)
- ADAMs as mediators of EGF receptor transactivation by G protein-coupled receptors.
Their papers
- Angiotensin II cell signaling: physiological and pathological effects in the cardiovascular system. (2007) — 1,953 citations