Reactive oxygen species in the vasculature: molecular and cellular mechanisms.

By Y. Taniyama, K. Griendling. HYPERTENSION, 2003

1,078 citations recorded by the source corpus.

Subfields: Biochemistry, Immunology, Physiology

View Paper: https://www.semanticscholar.org/paper/767365c7d7a486c9ae7c6ec8c0e02a16a7cef8af

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Biochemistry

1. E. Schiffrin
2. Rhian M. Touyz
3. K. Griendling
4. T. Vlachogianni
5. A. Valavanidis

In Immunology

1. Rhian M. Touyz
2. E. Schiffrin
3. P. Mehta
4. K. Griendling
5. S. Dikalov

In Physiology

1. E. Schiffrin
2. Rhian M. Touyz
3. K. Griendling
4. P. Mehta
5. T. Vlachogianni
6. Konstantinos Fiotakis
7. A. Valavanidis
8. Spyridon Loridas

Citations in the corpus

• Pulmonary Oxidative Stress, Inflammation and Cancer: Respirable Particulate Matter, Fibrous Dusts and Ozone as Major Causes of Lung Carcinogenesis through Reactive Oxygen Species Mechanisms (2013, background): “pulmonary region, may be susceptible for adverse health effects of PM inhalation [88].”
• Reactive oxygen species in vascular biology: implications in hypertension (2004, positive): “Other enzymatic sources capable of generating ROS in the vasculature are xanthine oxidase, cytochrome P450, mitochondrial respiratory chain enzymes, and phagocytederived myeloperoxidase (Taniyama and Griendling 2003; Fig.”
• Angiotensin II cell signaling: physiological and pathological effects in the cardiovascular system. (2007, background): “ANG II is a potent mediator of oxidative stress and oxidant signaling (186, 191, 201, 217).”