Leah Boyer

Neurology · 2009–2011

A researcher in the Science corpus: 2 papers; trusts 1 researcher and is trusted by 0 researchers here, as inferred from the citation graph.

Subfields

Whom Leah Boyer trusts

Their strongest outbound trust, strongest first, inferred from their citations.

  1. P. Whitton (in Cellular and Molecular Neuroscience)

Papers from Leah Boyer's perspective

Ordered by how strongly the researchers Leah Boyer trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Activation of innate immunity in the CNS triggers neurodegeneration through a Toll-like receptor 4-dependent pathway (Proceedings of the National Academy of Sciences of the United States of America)
  2. A SUMOylation-dependent pathway mediates transrepression of inflammatory response genes by PPAR-γ (Nature)
  3. Fibrils formed in vitro from alpha-synuclein and two mutant forms linked to Parkinson's disease are typical amyloid. (Biochemistry)
  4. Acceleration of oligomerization, not fibrillization, is a shared property of both alpha-synuclein mutations linked to early-onset Parkinson's disease: implications for pathogenesis and therapy. (Proceedings of the National Academy of Sciences of the United States of America)
  5. Molecular-level secondary structure, polymorphism, and dynamics of full-length alpha-synuclein fibrils studied by solid-state NMR. (Proceedings of the National Academy of Sciences of the United States of America)
  6. alpha-Synuclein locus triplication causes Parkinson's disease. (Science)
  7. Vesicle permeabilization by protofibrillar alpha-synuclein: implications for the pathogenesis and treatment of Parkinson's disease. (Biochemistry)
  8. A structural model for Alzheimer's beta -amyloid fibrils based on experimental constraints from solid state NMR. (Proceedings of the National Academy of Sciences of the United States of America)
  9. Alpha-synuclein and neurodegenerative diseases (Nature Reviews Neuroscience)
  10. Common Structure of Soluble Amyloid Oligomers Implies Common Mechanism of Pathogenesis (Science)

Their papers