A. Kong

Molecular Biology · 2008–2009

A researcher in the Science corpus: 2 papers; trusts 4 researchers and is trusted by 0 researchers here, as inferred from the citation graph.

Subfields

Whom A. Kong trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. Donna D. Zhang

Papers from A. Kong's perspective

Ordered by how strongly the researchers A. Kong trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Protection against electrophile and oxidant stress by induction of the phase 2 response: Fate of cysteines of the Keap1 sensor modified by inducers (Proceedings of the National Academy of Sciences of the United States of America)
  2. Regulatory mechanisms controlling gene expression mediated by the antioxidant response element. (Annual Review of Pharmacology and Toxicology)
  3. BTB Protein Keap1 Targets Antioxidant Transcription Factor Nrf2 for Ubiquitination by the Cullin 3-Roc1 Ligase (Molecular and Cellular Biology)
  4. Oxidative Stress Sensor Keap1 Functions as an Adaptor for Cul3-Based E3 Ligase To Regulate Proteasomal Degradation of Nrf2 (Molecular and Cellular Biology)
  5. Nrf2-Keap1 defines a physiologically important stress response mechanism. (Trends in Molecular Medicine)
  6. Keap1-null mutation leads to postnatal lethality due to constitutive Nrf2 activation (Nature Genetics)
  7. Nrf2 Is a Direct PERK Substrate and Effector of PERK-Dependent Cell Survival (Molecular and Cellular Biology)
  8. Keap1 Is a Redox-Regulated Substrate Adaptor Protein for a Cul3-Dependent Ubiquitin Ligase Complex (Molecular and Cellular Biology)
  9. Cellular mechanisms of redox cell signalling: role of cysteine modification in controlling antioxidant defences in response to electrophilic lipid oxidation products. (Biochemical Journal)
  10. The Keap1-BTB Protein Is an Adaptor That Bridges Nrf2 to a Cul3-Based E3 Ligase: Oxidative Stress Sensing by a Cul3-Keap1 Ligase (Molecular and Cellular Biology)

Their papers