Frédéric Dayan
Cancer Research · 2006–2009
A researcher in the Science corpus: 2 papers; trusts 1 researcher and is trusted by 3 researchers here, as inferred from the citation graph.
Subfields
- Cancer Research — 30 citations into it; #891 of 1,780 by trust here, #895 by citations
Whom Frédéric Dayan trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts Frédéric Dayan overall
Papers from Frédéric Dayan's perspective
Ordered by how strongly the researchers Frédéric Dayan trusts endorse each paper. Same corpus, a different order for every researcher.
- Inhibition of HIF is necessary for tumor suppression by the von Hippel-Lindau protein. (Cancer Cell)
- Why do cancers have high aerobic glycolysis? (Nature Reviews. Cancer)
- C. elegans EGL-9 and mammalian homologs define a family of dioxygenases that regulate HIF by prolyl hydroxylation. (Cell)
- Targeting HIF-1 for cancer therapy (Nature Reviews. Cancer)
- Hypoxia-inducible factor determines sensitivity to inhibitors of mTOR in kidney cancer (Nature Medicine)
- A Conserved Family of Prolyl-4-Hydroxylases That Modify HIF (Science)
- Expression of the gene encoding the proapoptotic Nip3 protein is induced by hypoxia. (Proceedings of the National Academy of Sciences of the United States of America)
- Epithelial-mesenchymal transitions in development and pathologies. (Current Opinion in Cell Biology)
- HIF prolyl‐hydroxylase 2 is the key oxygen sensor setting low steady‐state levels of HIF‐1α in normoxia (EMBO Journal)
- Role of VHL gene mutation in human cancer. (Journal of Clinical Oncology)