Satomi Miwa
Molecular Biology · 2004–2022
A researcher in the Science corpus: 4 papers; trusts 1 researcher and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 44 citations into it
Whom Satomi Miwa trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Papers from Satomi Miwa's perspective
Ordered by how strongly the researchers Satomi Miwa trusts endorse each paper. Same corpus, a different order for every researcher.
- Feedback between p21 and reactive oxygen production is necessary for cell senescence (Molecular Systems Biology)
- Mitochondrial Dysfunction Induces Senescence with a Distinct Secretory Phenotype (Cell Metabolism)
- Design and validation of a histological scoring system for nonalcoholic fatty liver disease (Hepatology)
- Mammalian Sir2 Homolog SIRT3 Regulates Global Mitochondrial Lysine Acetylation (Molecular and Cellular Biology)
- Senescence-messaging secretome: SMS-ing cellular stress (Nature Reviews. Cancer)
- The Achilles’ heel of senescent cells: from transcriptome to senolytic drugs (Aging Cell)
- Oncogene-induced senescence is a DNA damage response triggered by DNA hyper-replication (Nature)
- Mitochondrial Dysfunction Accounts for the Stochastic Heterogeneity in Telomere-Dependent Senescence (PLoS Biology)
- Oxidative stress shortens telomeres. (TIBS -Trends in Biochemical Sciences. Regular ed)
- Chronic senolytic treatment alleviates established vasomotor dysfunction in aged or atherosclerotic mice (Aging Cell)
Their papers
- Feedback between p21 and reactive oxygen production is necessary for cell senescence (2010) — 963 citations
- Mitochondrial superoxide: production, biological effects, and activation of uncoupling proteins. (2004) — 1,054 citations
- Cellular senescence drives age-dependent hepatic steatosis (2017) — 924 citations
- Mitochondrial dysfunction in cell senescence and aging (2022) — 787 citations