Sérgio T. Ferreira
Physiology · 2007–2019
A researcher in the Science corpus: 5 papers; trusts 5 researchers and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Physiology — 69 citations into it
- Molecular Biology — 47 citations into it
- Cellular and Molecular Neuroscience — 32 citations into it
- Epidemiology — 23 citations into it
- Pharmacology — 18 citations into it
- Surgery — 12 citations into it
Whom Sérgio T. Ferreira trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Papers from Sérgio T. Ferreira's perspective
Ordered by how strongly the researchers Sérgio T. Ferreira trusts endorse each paper. Same corpus, a different order for every researcher.
- Mechanism by which high-dose aspirin improves glucose metabolism in type 2 diabetes. (Journal of Clinical Investigation)
- Phosphorylation of Ser307 in Insulin Receptor Substrate-1 Blocks Interactions with the Insulin Receptor and Inhibits Insulin Action* (Journal of Biological Chemistry)
- Pathways towards and away from Alzheimer's disease (Nature)
- Chronic inflammation in fat plays a crucial role in the development of obesity-related insulin resistance. (Journal of Clinical Investigation)
- Defects in IGF-1 receptor, insulin receptor and IRS-1/2 in Alzheimer's disease indicate possible resistance to IGF-1 and insulin signalling. (Neurobiology of Aging)
- Naturally secreted oligomers of amyloid β protein potently inhibit hippocampal long-term potentiation in vivo (Nature)
- Increased risk of type 2 diabetes in Alzheimer disease. (Diabetes)
- Intranasal insulin improves cognition and modulates β-amyloid in early AD (Neurology)
- Regulation of NMDA receptor trafficking by amyloid-β (Nature Neuroscience)
- Efficacy of rosiglitazone in a genetically defined population with mild-to-moderate Alzheimer's disease (The Pharmacogenomics Journal)
Their papers
- Exercise-linked FNDC5/irisin rescues synaptic plasticity and memory defects in Alzheimer’s models (2019) — 801 citations
- Inflammation, Defective Insulin Signaling, and Mitochondrial Dysfunction as Common Molecular Denominators Connecting Type 2 Diabetes to Alzheimer Disease (2014) — 606 citations
- An anti-diabetes agent protects the mouse brain from defective insulin signaling caused by Alzheimer's disease- associated Aβ oligomers. (2012) — 813 citations
- Protection of synapses against Alzheimer's-linked toxins: Insulin signaling prevents the pathogenic binding of Aβ oligomers (2009) — 616 citations
- Aβ Oligomers Induce Neuronal Oxidative Stress through an N-Methyl-D-aspartate Receptor-dependent Mechanism That Is Blocked by the Alzheimer Drug Memantine* (2007) — 904 citations