David B. Teplow

Physiology · 2001–2009

A researcher in the Science corpus: 7 papers; trusts 8 researchers and is trusted by 2 researchers here, as inferred from the citation graph.

Subfields

• Physiology — 43 citations into it; #1,586 of 2,276 by trust here, #543 by citations
• Molecular Biology — 22 citations into it; #14,393 of 17,773 by trust here, #6,095 by citations
• Pharmacology — 18 citations into it
• Neurology — 16 citations into it
• Psychiatry and Mental health — 5 citations into it

Whom David B. Teplow trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

1. M. Kirkitadze
2. W. Klein
3. D. Selkoe
4. D. Walsh

Who trusts David B. Teplow overall

1. G. Bitan
2. M. Kirkitadze

Papers from David B. Teplow's perspective

Ordered by how strongly the researchers David B. Teplow trusts endorse each paper. Same corpus, a different order for every researcher.

1. Targeting small Aβ oligomers: the solution to an Alzheimer's disease conundrum?
2. Formation of Neurofibrillary Tangles in P301L Tau Transgenic Mice Induced by Aβ42 Fibrils (Science)
3. Naturally secreted oligomers of amyloid β protein potently inhibit hippocampal long-term potentiation in vivo (Nature)
4. Peripheral anti-Aβ antibody alters CNS and plasma Aβ clearance and decreases brain Aβ burden in a mouse model of Alzheimer's disease (Proceedings of the National Academy of Sciences of the United States of America)
5. Intraneuronal Aβ42 Accumulation in Human Brain
6. Neuropathology of Alzheimer’s Disease
7. Tau is essential to β-amyloid-induced neurotoxicity (Proceedings of the National Academy of Sciences of the United States of America)
8. Aggresomes, inclusion bodies and protein aggregation. (Trends in Cell Biology)
9. Treatment with a Copper-Zinc Chelator Markedly and Rapidly Inhibits β-Amyloid Accumulation in Alzheimer's Disease Transgenic Mice
10. Correlation between elevated levels of amyloid beta-peptide in the brain and cognitive decline. (Journal of the American Medical Association (JAMA))

Their papers

• Identification and characterization of key kinetic intermediates in amyloid beta-protein fibrillogenesis. (2001) — 651 citations
• The 'Arctic' APP mutation (E693G) causes Alzheimer's disease by enhanced Aβ protofibril formation (2001) — 1,244 citations
• Paradigm shifts in Alzheimer's disease and other neurodegenerative disorders: The emerging role of oligomeric assemblies (2002) — 595 citations
• Structure–neurotoxicity relationships of amyloid β-protein oligomers (2009) — 759 citations
• Amyloid-β protein oligomerization and the importance of tetramers and dodecamers in the aetiology of Alzheimer’s disease (2009) — 839 citations
• In silico study of amyloid β-protein folding and oligomerization (2004) — 311 citations
• Presenilin‐dependent γ‐secretase processing of β‐amyloid precursor protein at a site corresponding to the S3 cleavage of Notch (2001) — 460 citations