Martin R. G. Taylor
Molecular Biology · 2012
A researcher in the Science corpus: 1 paper; trusts 1 researcher and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 17 citations into it
Whom Martin R. G. Taylor trusts
Their strongest outbound trust, strongest first, inferred from their citations.
- J. Bartek (in Molecular Biology)
Papers from Martin R. G. Taylor's perspective
Ordered by how strongly the researchers Martin R. G. Taylor trusts endorse each paper. Same corpus, a different order for every researcher.
- DNA end resection, homologous recombination and DNA damage checkpoint activation require CDK1 (Nature)
- The Bloom's syndrome helicase suppresses crossing over during homologous recombination (Nature)
- Structural basis for the methylation state-specific recognition of histone H4-K20 by 53BP1 and Crb2 in DNA repair. (Cell)
- 53BP1 loss rescues BRCA1 deficiency and is associated with triple-negative and BRCA-mutated breast cancers (Nature Structural &Molecular Biology)
- RAP80 Targets BRCA1 to Specific Ubiquitin Structures at DNA Damage Sites (Science)
- An ATM-Dependent Transcriptional Silencing Program is Transmitted Through Chromatin in Cis to DNA Double Strand Breaks (Cell)
- 53BP1 inhibits homologous recombination in Brca1-deficient cells by blocking resection of DNA breaks (Cell)
- The RING Heterodimer BRCA1-BARD1 Is a Ubiquitin Ligase Inactivated by a Breast Cancer-derived Mutation* (Journal of Biological Chemistry)
- Mechanism of eukaryotic homologous recombination. (Annual Review of Biochemistry)
- PALB2 is an integral component of the BRCA complex required for homologous recombination repair (Proceedings of the National Academy of Sciences of the United States of America)
Their papers
- Playing the end game: DNA double-strand break repair pathway choice. (2012) — 1,608 citations