J. Millet
Infectious Diseases · 2012–2016
A researcher in the Science corpus: 4 papers; trusts 3 researchers and is trusted by 10 researchers here, as inferred from the citation graph.
Subfields
- Infectious Diseases — 29 citations into it; #51 of 589 by trust here, #142 by citations
- Animal Science and Zoology — 13 citations into it; #40 of 476 by trust here, #48 by citations
- Genetics — 5 citations into it; #5,448 of 6,854 by trust here, #5,952 by citations
Whom J. Millet trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts J. Millet overall
- A. Walls
- Fang Li
- M. A. Tortorici
- Gary R. Whittaker
- A. Tortoricia
- Young-Jun Park
- X. Xiong
- J. Shang
- D. Veesler
- David Veeslera
Papers from J. Millet's perspective
Ordered by how strongly the researchers J. Millet trusts endorse each paper. Same corpus, a different order for every researcher.
- What’s new in the renin-angiotensin system?
- Coronaviruses post-SARS: Update on replication and pathogenesis (Nature Reviews Microbiology)
- Genomic Characterization of a Newly Discovered Coronavirus Associated with Acute Respiratory Distress Syndrome in Humans (mBio)
- The Coronavirus Spike Protein Is a Class I Virus Fusion Protein: Structural and Functional Characterization of the Fusion Core Complex (Journal of Virology)
- Receptor and viral determinants of SARS-coronavirus adaptation to human ACE2 (EMBO Journal)
- Infectious Hepatitis C Virus Pseudo-particles Containing Functional E1–E2 Envelope Protein Complexes (Journal of Experimental Medicine)
- The biology and therapeutic targeting of the proprotein convertases (Nature reviews. Drug discovery)
- Activation of the SARS coronavirus spike protein via sequential proteolytic cleavage at two distinct sites (Proceedings of the National Academy of Sciences of the United States of America)
- Inhibitors of cathepsin L prevent severe acute respiratory syndrome coronavirus entry. (Proceedings of the National Academy of Sciences of the United States of America)
- Evidence that TMPRSS2 Activates the Severe Acute Respiratory Syndrome Coronavirus Spike Protein for Membrane Fusion and Reduces Viral Control by the Humoral Immune Response (Journal of Virology)
Their papers
- Murine Leukemia Virus (MLV)-based Coronavirus Spike-pseudotyped Particle Production and Infection (2016) — 97 citations
- Host cell entry of Middle East respiratory syndrome coronavirus after two-step, furin-mediated activation of the spike protein (2014) — 684 citations
- Host cell proteases: critical determinants of coronavirus tropism and pathogenesis (2014) — 857 citations
- Mechanisms of Coronavirus Cell Entry Mediated by the Viral Spike Protein (2012) — 1,279 citations