E. Mills
Immunology · Cell · 2014–2018
A researcher in the Science corpus: 4 papers; trusts 1 researcher and is trusted by 1 researcher here, as inferred from the citation graph.
Subfields
- Immunology — 53 citations into it; #1,816 of 2,160 by trust here, #1,186 by citations
Whom E. Mills trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts E. Mills overall
Papers from E. Mills's perspective
Ordered by how strongly the researchers E. Mills trusts endorse each paper. Same corpus, a different order for every researcher.
- Mitochondria are required for antigen-specific T cell activation through reactive oxygen species signaling (Immunity)
- HTSeq—a Python framework to work with high-throughput sequencing data (bioRxiv)
- HIF overexpression correlates with biallelic loss of fumarate hydratase in renal cancer: novel role of fumarate in regulation of HIF stability. (Cancer Cell)
- Inhibition of α-KG-dependent histone and DNA demethylases by fumarate and succinate that are accumulated in mutations of FH and SDH tumor suppressors. (Genes & Development)
- Ischaemic accumulation of succinate controls reperfusion injury through mitochondrial ROS (Nature)
- Network integration of parallel metabolic and transcriptional data reveals metabolic modules that regulate macrophage polarization. (Immunity)
- Reactive Oxygen Species Generated at Mitochondrial Complex III Stabilize Hypoxia-inducible Factor-1α during Hypoxia (Journal of Biological Chemistry)
- Ultrafast and memory-efficient alignment of short DNA sequences to the human genome (Genome Biology)
- Selective Targeting of a Redox-active Ubiquinone to Mitochondria within Cells (Journal of Biological Chemistry)
- Immunometabolism governs dendritic cell and macrophage function (Journal of Experimental Medicine)
Their papers
- Repurposing mitochondria from ATP production to ROS generation drives a pro-inflammatory phenotype in macrophages that depends on succinate oxidation by complex II (2016) — 1,997 citations
- Itaconate is an anti-inflammatory metabolite that activates Nrf2 via alkylation of KEAP1 (2018) — 1,634 citations
- Mitochondria are the powerhouses of immunity (2017) — 1,002 citations
- Succinate: a metabolic signal in inflammation. (2014) — 657 citations