P. Shaw

Neurology · Annals of Neurology · 2006–2022

A researcher in the Science corpus: 9 papers; trusts 6 researchers and is trusted by 0 researchers here, as inferred from the citation graph.

Subfields

Whom P. Shaw trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. Christopher A. Ross
  2. M. Beal

Papers from P. Shaw's perspective

Ordered by how strongly the researchers P. Shaw trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Ubiquitinated TDP-43 in Frontotemporal Lobar Degeneration and Amyotrophic Lateral Sclerosis (Science)
  2. TARDBP mutations in individuals with sporadic and familial amyotrophic lateral sclerosis (Nature Genetics)
  3. Nuclear factor TDP‐43 and SR proteins promote in vitro and in vivo CFTR exon 9 skipping (EMBO Journal)
  4. TDP-43 is a component of ubiquitin-positive tau-negative inclusions in frontotemporal lobar degeneration and amyotrophic lateral sclerosis. (Biochemical and Biophysical Research Communications - BBRC)
  5. Protofibrils, pores, fibrils, and neurodegeneration: separating the responsible protein aggregates from the innocent bystanders. (Annual Review of Neuroscience)
  6. Neuropathologic diagnostic and nosologic criteria for frontotemporal lobar degeneration: consensus of the Consortium for Frontotemporal Lobar Degeneration (Acta Neuropathologica)
  7. Molecular biology of amyotrophic lateral sclerosis: insights from genetics (Nature Reviews Neuroscience)
  8. Mutated Human SOD1 Causes Dysfunction of Oxidative Phosphorylation in Mitochondria of Transgenic Mice* (Journal of Biological Chemistry)
  9. Rethinking ALS: the FUS about TDP-43. (Cell)
  10. Astrocytes expressing ALS-linked mutated SOD1 release factors selectively toxic to motor neurons (Nature Neuroscience)

Their papers