Deepak Nagrath

Molecular Biology · 2016–2017

A researcher in the Science corpus: 2 papers; trusts 2 researchers and is trusted by 0 researchers here, as inferred from the citation graph.

Subfields

Whom Deepak Nagrath trusts

Their strongest outbound trust, strongest first, inferred from their citations.

  1. Craig B. Thompson (in Cancer Research)
  2. G. Semenza (in Cancer Research)

Papers from Deepak Nagrath's perspective

Ordered by how strongly the researchers Deepak Nagrath trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Beyond aerobic glycolysis: Transformed cells can engage in glutamine metabolism that exceeds the requirement for protein and nucleotide synthesis (Proceedings of the National Academy of Sciences of the United States of America)
  2. Mitochondrial metabolism and ROS generation are essential for Kras-mediated tumorigenicity (Proceedings of the National Academy of Sciences of the United States of America)
  3. The common feature of leukemia-associated IDH1 and IDH2 mutations is a neomorphic enzymatic activity that converts α-ketoglutarate to 2-hydroxyglutarate (Cancer Cell)
  4. Myc regulates a transcriptional program that stimulates mitochondrial glutaminolysis and leads to glutamine addiction (Proceedings of the National Academy of Sciences of the United States of America)
  5. Cancer-associated IDH1 mutations produce 2-hydroxyglutarate (Nature)
  6. Faculty Opinions recommendation of Oncogenic Kras maintains pancreatic tumors through regulation of anabolic glucose metabolism.
  7. Hallmarks of cancer: the next generation. (Cell)
  8. Glutamine supports pancreatic cancer growth through a Kras-regulated metabolic pathway (Nature)
  9. IDH1 and IDH2 mutations in gliomas. (New England Journal of Medicine)
  10. Targeting mitochondrial glutaminase activity inhibits oncogenic transformation (Cancer Cell)

Their papers