L. Lankhorst
Physiology · 2022
A researcher in the Science corpus: 1 paper; trusts 1 researcher and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Physiology — 50 citations into it
Whom L. Lankhorst trusts
Their strongest outbound trust, strongest first, inferred from their citations.
- J. Campisi (in Physiology)
Papers from L. Lankhorst's perspective
Ordered by how strongly the researchers L. Lankhorst trusts endorse each paper. Same corpus, a different order for every researcher.
- Senescence and tumour clearance is triggered by p53 restoration in murine liver carcinomas (Nature)
- Chemokine signaling via the CXCR2 receptor reinforces senescence. (Cell)
- Senescence of activated stellate cells limits liver fibrosis (Cell)
- Plasminogen activator inhibitor-1 is a critical downstream target of p53 in the induction of replicative senescence (Nature Cell Biology)
- Senescent fibroblasts promote epithelial cell growth and tumorigenesis: A link between cancer and aging (Proceedings of the National Academy of Sciences of the United States of America)
- Senescence-Associated Secretory Phenotypes Reveal Cell-Nonautonomous Functions of Oncogenic RAS and the p53 Tumor Suppressor (PLoS Biology)
- Stromal-epithelial interactions in aging and cancer: senescent fibroblasts alter epithelial cell differentiation (Journal of Cell Science)
- Rb-mediated heterochromatin formation and silencing of E2F target genes during cellular senescence. (Cell)
- Tumour biology: Senescence in premalignant tumours (Nature)
- Oncogene-induced senescence is a DNA damage response triggered by DNA hyper-replication (Nature)
Their papers
- Exploiting senescence for the treatment of cancer (2022) — 691 citations