Ann M. Bode
Oncology · 2004
A researcher in the Science corpus: 1 paper; trusts 1 researcher and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Oncology — 17 citations into it
Whom Ann M. Bode trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts Ann M. Bode overall
Papers from Ann M. Bode's perspective
Ordered by how strongly the researchers Ann M. Bode trusts endorse each paper. Same corpus, a different order for every researcher.
- Deacetylation of p53 modulates its effect on cell growth and apoptosis (Nature)
- Post-translational modifications and activation of p53 by genotoxic stresses. (European Journal of Biochemistry)
- p300/CBP‐mediated p53 acetylation is commonly induced by p53‐activating agents and inhibited by MDM2 (EMBO Journal)
- Acetylation of p53 activates transcription through recruitment of coactivators/histone acetyltransferases. (Molecules and Cells)
- Mdm2-mediated NEDD8 conjugation of p53 inhibits its transcriptional activity. (Cell)
- Human SIR2 deacetylates p53 and antagonizes PML/p53‐induced cellular senescence (EMBO Journal)
- Mono- Versus Polyubiquitination: Differential Control of p53 Fate by Mdm2 (Science)
- Surfing the p53 network (Nature)
- hSIR2(SIRT1) functions as an NAD-dependent p53 deacetylase. (Cell)
- The human homologs of checkpoint kinases Chk1 and Cds1 (Chk2) phosphorylate p53 at multiple DNA damage-inducible sites. (Genes & Development)
Their papers
- Post-translational modification of p53 in tumorigenesis (2004) — 1,315 citations