M. J. Saad
Epidemiology · 2005–2009
A researcher in the Science corpus: 3 papers; trusts 1 researcher and is trusted by 1 researcher here, as inferred from the citation graph.
Subfields
- Epidemiology — 26 citations into it; #1,751 of 2,148 by trust here, #806 by citations
- Immunology — 13 citations into it
- Physiology — 13 citations into it
Whom M. J. Saad trusts
Their strongest outbound trust, strongest first, inferred from their citations.
- G. Hotamisligil (in Epidemiology)
Who trusts M. J. Saad
- G. Hotamışlıgil (in Epidemiology)
Papers from M. J. Saad's perspective
Ordered by how strongly the researchers M. J. Saad trusts endorse each paper. Same corpus, a different order for every researcher.
- Endoplasmic Reticulum Stress Links Obesity, Insulin Action, and Type 2 Diabetes (Science)
- Central nervous system control of food intake (Nature)
- Phosphorylation of Ser307 in Insulin Receptor Substrate-1 Blocks Interactions with the Insulin Receptor and Inhibits Insulin Action* (Journal of Biological Chemistry)
- SOCS-1 and SOCS-3 Block Insulin Signaling by Ubiquitin-mediated Degradation of IRS1 and IRS2* (Journal of Biological Chemistry)
- TLR4 links innate immunity and fatty acid-induced insulin resistance. (Journal of Clinical Investigation)
- Region-specific leptin resistance within the hypothalamus of diet-induced obese mice. (Endocrinology)
- Anti-inflammatory cyclopentenone prostaglandins are direct inhibitors of IκB kinase (Nature)
- Inflammatory pathways and insulin action (International Journal of Obesity)
- A central role for JNK in obesity and insulin resistance (Nature)
- Serine Phosphorylation of Insulin Receptor Substrate 1 by Inhibitor κB Kinase Complex* 210 (Journal of Biological Chemistry)
Their papers
- Saturated Fatty Acids Produce an Inflammatory Response Predominantly through the Activation of TLR4 Signaling in Hypothalamus: Implications for the Pathogenesis of Obesity (2009) — 1,005 citations
- Loss-of-Function Mutation in Toll-Like Receptor 4 Prevents Diet-Induced Obesity and Insulin Resistance (2007) — 835 citations
- Consumption of a fat-rich diet activates a proinflammatory response and induces insulin resistance in the hypothalamus. (2005) — 1,102 citations