K. Ashe
Physiology · 2000–2012
A researcher in the Science corpus: 11 papers; trusts 1 researcher and is trusted by 2 researchers here, as inferred from the citation graph.
Subfields
- Physiology — 48 citations into it; #1,096 of 2,276 by trust here, #121 by citations
- Cellular and Molecular Neuroscience — 27 citations into it
- Computational Theory and Mathematics — 11 citations into it; #11,307 of 12,182 by trust here, #1,101 by citations
Whom K. Ashe trusts
Their strongest outbound trust, strongest first, inferred from their citations.
- D. Selkoe (in Physiology)
Who trusts K. Ashe
Papers from K. Ashe's perspective
Ordered by how strongly the researchers K. Ashe trusts endorse each paper. Same corpus, a different order for every researcher.
- A beta peptide vaccination prevents memory loss in an animal model of Alzheimer's disease. (Nature)
- Neuropathological stageing of Alzheimer-related changes
- Alzheimer's Disease Is a Synaptic Failure (Science)
- Oligomeric amyloid β associates with postsynaptic densities and correlates with excitatory synapse loss near senile plaques (Proceedings of the National Academy of Sciences of the United States of America)
- Age-Dependent Changes in Brain, CSF, and Plasma Amyloid β Protein in the Tg2576 Transgenic Mouse Model of Alzheimer's Disease (Journal of Neuroscience)
- Memory from A to Z: Keywords, Concepts, and Beyond
- Altered expression of synaptic proteins occurs early during progression of Alzheimer’s disease (Neurology)
- Cerebral metabolic and cognitive decline in persons at genetic risk for Alzheimer's disease. (Proceedings of the National Academy of Sciences of the United States of America)
- Patterns of brain activation in people at risk for Alzheimer's disease. (New England Journal of Medicine)
- High-Level Neuronal Expression of Aβ1–42 in Wild-Type Human Amyloid Protein Precursor Transgenic Mice: Synaptotoxicity without Plaque Formation (Journal of Neuroscience)
Their papers
- Natural oligomers of the amyloid-β protein specifically disrupt cognitive function (2005) — 1,747 citations
- The Relationship between Aβ and Memory in the Tg2576 Mouse Model of Alzheimer's Disease (2002) — 653 citations
- Age-Dependent Changes in Brain, CSF, and Plasma Amyloid β Protein in the Tg2576 Transgenic Mouse Model of Alzheimer's Disease (2001) — 926 citations
- Propagation of tau pathology in a model of early Alzheimer’s disease (2012) — 1,415 citations
- Tau mislocalization to dendritic spines mediates synaptic dysfunction independently of neurodegeneration (2010) — 1,045 citations
- A specific amyloid-beta protein assembly in the brain impairs memory. (2006) — 1,267 citations
- A specific amyloid-β protein assembly in the brain impairs memory (2006) — 2,628 citations
- Tau Suppression in a Neurodegenerative Mouse Model Improves Memory Function (2005) — 1,772 citations
- Age-Dependent Neurofibrillary Tangle Formation, Neuron Loss, and Memory Impairment in a Mouse Model of Human Tauopathy (P301L) (2005) — 676 citations
- Docosahexaenoic acid protects from dendritic pathology in an Alzheimer's disease mouse model. (2004) — 747 citations
- Ibuprofen Suppresses Plaque Pathology and Inflammation in a Mouse Model for Alzheimer's Disease (2000) — 946 citations