T. Shibata

Molecular Biology · with Kang · 2006

A researcher in the Science corpus: 1 paper; trusts 1 researcher and is trusted by 0 researchers here, as inferred from the citation graph.

Subfields

Whom T. Shibata trusts

Their strongest outbound trust, strongest first, inferred from their citations.

  1. Masayuki Yamamoto (in Molecular Biology)

Papers from T. Shibata's perspective

Ordered by how strongly the researchers T. Shibata trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Keap1 regulates both cytoplasmic‐nuclear shuttling and degradation of Nrf2 in response to electrophiles (Genes to Cells)
  2. Oxidative Stress Sensor Keap1 Functions as an Adaptor for Cul3-Based E3 Ligase To Regulate Proteasomal Degradation of Nrf2 (Molecular and Cellular Biology)
  3. Structure of the Cul1–Rbx1–Skp1–F boxSkp2 SCF ubiquitin ligase complex (Nature)
  4. Direct evidence that sulfhydryl groups of Keap1 are the sensors regulating induction of phase 2 enzymes that protect against carcinogens and oxidants (Proceedings of the National Academy of Sciences of the United States of America)
  5. Regulatory mechanisms controlling gene expression mediated by the antioxidant response element. (Annual Review of Pharmacology and Toxicology)
  6. BTB Protein Keap1 Targets Antioxidant Transcription Factor Nrf2 for Ubiquitination by the Cullin 3-Roc1 Ligase (Molecular and Cellular Biology)
  7. Distinct Cysteine Residues in Keap1 Are Required for Keap1-Dependent Ubiquitination of Nrf2 and for Stabilization of Nrf2 by Chemopreventive Agents and Oxidative Stress (Molecular and Cellular Biology)
  8. Protection against electrophile and oxidant stress by induction of the phase 2 response: Fate of cysteines of the Keap1 sensor modified by inducers (Proceedings of the National Academy of Sciences of the United States of America)
  9. Potency of Michael reaction acceptors as inducers of enzymes that protect against carcinogenesis depends on their reactivity with sulfhydryl groups (Proceedings of the National Academy of Sciences of the United States of America)
  10. Nrf2-Keap1 defines a physiologically important stress response mechanism. (Trends in Molecular Medicine)

Their papers