J. M. García-Martínez
Molecular Biology · 2008
A researcher in the Science corpus: 1 paper; trusts 1 researcher and is trusted by 4 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 21 citations into it; #7,638 of 17,773 by trust here, #11,818 by citations
Whom J. M. García-Martínez trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Who trusts J. M. García-Martínez overall
Papers from J. M. García-Martínez's perspective
Ordered by how strongly the researchers J. M. García-Martínez trusts endorse each paper. Same corpus, a different order for every researcher.
- Phosphorylation and Regulation of Akt/PKB by the Rictor-mTOR Complex (Science)
- Two TOR complexes, only one of which is rapamycin sensitive, have distinct roles in cell growth control. (Molecules and Cells)
- mTOR interacts with raptor to form a nutrient-sensitive complex that signals to the cell growth machinery. (Cell)
- Phosphorylation and functional inactivation of TSC2 by Erk implications for tuberous sclerosis and cancer pathogenesis. (Cell)
- Rictor, a novel binding partner of mTOR, defines a rapamycin-insensitive and raptor-independent pathway that regulates the cytoskeleton. (Current Biology)
- TSC2 mediates cellular energy response to control cell growth and survival. (Cell)
- mTOR·RICTOR Is the Ser473 Kinase for Akt/Protein Kinase B in 3T3-L1 Adipocytes* (Journal of Biological Chemistry)
- The TSC1-2 tumor suppressor controls insulin–PI3K signaling via regulation of IRS proteins (Journal of Cell Biology)
- Raptor, a binding partner of target of rapamycin (TOR), mediates TOR action. (Cell)
- Tsc tumour suppressor proteins antagonize amino-acid–TOR signalling (Nature Cell Biology)