M. O’Driscoll

Molecular Biology · Nature Genetics · 2003–2012

A researcher in the Science corpus: 5 papers; trusts 2 researchers and is trusted by 3 researchers here, as inferred from the citation graph.

Subfields

• Molecular Biology — 52 citations into it; #9,889 of 17,773 by trust here, #5,655 by citations
• Oncology — 23 citations into it

Whom M. O’Driscoll trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

1. S. West
2. Stephen P. Jackson
3. T. Paull
4. O. Fernández-Capetillo
5. J. Falck

Who trusts M. O’Driscoll overall

1. F. Grin
2. H. Reinhardt
3. L. Marácz
4. N. Pokorn
5. D. Meek
6. Adrian C. Begg
7. Aaron Aslanian

Papers from M. O’Driscoll's perspective

Ordered by how strongly the researchers M. O’Driscoll trusts endorse each paper. Same corpus, a different order for every researcher.

1. Review Cancer Susceptibility and the Functions of BRCA1 and BRCA2
2. ATR and ATRIP: Partners in Checkpoint Signaling (Science)
3. Insights into DNA recombination from the structure of a RAD51–BRCA2 complex (Nature)
4. ATR disruption leads to chromosomal fragmentation and early embryonic lethality. (Genes & Development)
5. Genomic Instability in Mice Lacking Histone H2AX (Science)
6. A critical role for histone H2AX in recruitment of repair factors to nuclear foci after DNA damage. (Current Biology)
7. Targeting the DNA repair defect in BRCA mutant cells as a therapeutic strategy (Nature)
8. Histone H2AX phosphorylation is dispensable for the initial recognition of DNA breaks (Nature Cell Biology)
9. DNA damage-induced G2–M checkpoint activation by histone H2AX and 53BP1 (Nature Cell Biology)
10. MDC1 is a mediator of the mammalian DNA damage checkpoint (Nature)

Their papers

• ATM and DNA-PK Function Redundantly to Phosphorylate H2AX after Exposure to Ionizing Radiation (2004) — 1,057 citations
• A splicing mutation affecting expression of ataxia–telangiectasia and Rad3–related protein (ATR) results in Seckel syndrome (2003) — 855 citations
• The role of double-strand break repair — insights from human genetics (2006) — 611 citations
• Mutations in ORC1, encoding the largest subunit of the origin recognition complex, cause microcephalic primordial dwarfism resembling Meier-Gorlin syndrome (2011) — 224 citations
• De novo germline and postzygotic mutations in AKT3, PIK3R2 and PIK3CA cause a spectrum of related megalencephaly syndromes (2012) — 688 citations