E. Thies

Physiology · 2002–2010

A researcher in the Science corpus: 2 papers; trusts 2 researchers and is trusted by 3 researchers here, as inferred from the citation graph.

Subfields

• Physiology — 20 citations into it; #862 of 2,276 by trust here, #1,080 by citations
• Molecular Biology — 15 citations into it

Whom E. Thies trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

1. G. Burnstock

Who trusts E. Thies overall

1. R. Vale
2. E. Mandelkow

Papers from E. Thies's perspective

Ordered by how strongly the researchers E. Thies trusts endorse each paper. Same corpus, a different order for every researcher.

1. Enhanced Neurofibrillary Degeneration in Transgenic Mice Expressing Mutant Tau and APP (Science)
2. Common Structure of Soluble Amyloid Oligomers Implies Common Mechanism of Pathogenesis (Science)
3. Tauopathy in Drosophila: Neurodegeneration Without Neurofibrillary Tangles (Science)
4. S3‐03‐05: Transmission and Spreading of Tauopathy in Transgenic Mouse Brain
5. Cargo of Kinesin Identified as Jip Scaffolding Proteins and Associated Signaling Molecules (Journal of Cell Biology)
6. Structural Classification of Toxic Amyloid Oligomers* (Journal of Biological Chemistry)
7. Tau-mediated neurodegeneration in Alzheimer's disease and related disorders (Nature Reviews Neuroscience)
8. Tau blocks traffic of organelles, neurofilaments, and APP vesicles in neurons and enhances oxidative stress (Journal of Cell Biology)
9. A specific amyloid-beta protein assembly in the brain impairs memory. (Nature)
10. A Transcriptively Active Complex of APP with Fe65 and Histone Acetyltransferase Tip60 (Science)

Their papers

• Aβ Oligomers Cause Localized Ca2+ Elevation, Missorting of Endogenous Tau into Dendrites, Tau Phosphorylation, and Destruction of Microtubules and Spines (2010) — 656 citations
• Tau blocks traffic of organelles, neurofilaments, and APP vesicles in neurons and enhances oxidative stress (2002) — 919 citations